- Safe Gluten-Free Food List (Safe Ingredients)
- Unsafe Gluten-Free Food List (Unsafe Ingredients)
- Gluten-Free Alcoholic Beverages
- Celiac Disease Symptoms
- The Gluten-Free Diet 101 - A Beginner's Guide to Going Gluten-Free
- Interpretation of Celiac Disease Blood Test Results
- Is Buckwheat Flour Really Gluten-Free?
- Celiac Disease & Gluten Intolerance Research
Celiac Disease & Gluten Intolerance Research
Sufferers of clostridium difficile infection have new hope in fecal transplants. Doctors understand better why they work.
A new study looks at intestinal cell damage and systemic immune activation in individuals reporting wheat sensitivity, but no celiac disease.
Does the season or region of birth influence celiac disease risk? New science says yes.
Although serological tests are useful for identifying celiac disease, it is well known that a small minority of celiacs are seronegative, and show no blood markers for celiac disease. A team of researchers wanted to define the prevalence and features of seronegative compared to seropositive celiac disease, and to establish whether celiac disease is a common cause of seronegative villous atrophy.
Many doctors hear from celiac patients who suffer from persistent symptoms despite a long-term gluten-free diet. A research team recently set out to investigate the prevalence and severity of these symptoms in patients with variable duration of a gluten-free diet.
Study shows previously unappreciated diversity and plasticity of innate IEL compartment, along with loss of differentiation potential in patients with RCDII.
Some researchers have suggested that gluten may not be the actual trigger of symptoms in non-celiac gluten sensitivity. Others feel that gluten is definitely the trigger, especially in certain cases.
Metagenomics shows dysbiosis and a potentially pathogenic N. flavescens strain in adults with celiac disease.
Symptoms and damage in celiac disease is caused by partially-degraded gluten peptides from wheat, barley and rye. Susceptibility genes are necessary to trigger celiac disease, but they can't do it alone. Some researchers suspect that these susceptibility genes might get help from conditions resulting from unfavorable changes in the microbiota.
Study shows gliadin peptide p31-43 activates celiac-related innate immune pathways in vivo, also hints at a common mechanism for interaction between dietary gluten and viral infections in triggering celiac disease.
A gluten-free diet reverses type 1 innate lymphoid cells in the rectal mucosa of patients with non-celiac wheat sensitivity
Can aptamers help researchers design biosensors for fast and selective measurement of gluten in foods?
With funds from the German Research Foundation, the Universities of Mainz and Hohenheim join forces to study wheat sensitivity in search for better tolerated wheat varieties.
Doctors generally recommend that celiac disease patients receive pneumococcal vaccination, but little has been done to quantify risk levels.
Unique case details clinical celiac disease following duodenal switch surgery.
Do people with non-celiac wheat sensitivity have more anti-nuclear antibodies than those with celiac disease?
Do all patients with potential celiac disease need a gluten-free diet? The transformation of potential celiac disease to full-blown celiac disease has been described in some western clinical studies, but there is no good data on cases in Asia.
People with potential celiac disease have blood and genetic markers for celiac disease, but show little or no damage to the small intestinal mucosa. Do they always need a gluten-free diet?
Does the preferential xxpression of HLA-DQ2.5 Genes in celiac disease impact T cell response?
Considering the new ESPGHAN and BSPGHAN guidelines, how useful is celiac diagnosis without biopsy?