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Celiac Disease & Gluten-Free Diet Forums

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Celiac Disease & Gluten-Free Diet Blogs

  • kareng's Blog
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  • An Unmistakeable Journey
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  • My tummy used to hurt....
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  • The Patient Celiac
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  • Trials and Tribulations
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  • Cee Cee's Blog
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  • learning2cope's Blog
  • Research on South African Celiac Tours
  • lindylynn's Blog
  • Celiaction's Blog
  • shelly184's Blog
  • Melissa.77's Blog
  • Keating's Not-so-Glutenfree life
  • AmandasMommy's Blog
  • Coeliac, or just plain unlucky?
  • bandanamama's Blog
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  • Ellenor Whitty's Blog
  • Mama Me Gluten Free
  • Ohmyword's Blog
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  • Bear with me's Blog
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  • Scott's Celiac Blog
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  • Gluten Freedom
  • Angie Baker
  • Kimberly's Blog
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  • Shelby
  • Reinhard1's Blog
  • Silly Yak 08's Blog
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  • NotMollyRingwald's Blog
  • Searchin for a Primary Care Dr. In Redlands That is Knowledgeable about Celiac disease
  • num1habsfan's Blog
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  • Celiac-Positive
  • Jason's Mommy's Blog
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  • Lauren Johnson's Celiac Blog
  • I love my plant Cactus <3
  • Chele's Blog
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  • Blues Boulevard
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  • Inspiration
  • Cindy Neshe's Blog
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  • Jema's Blog
  • What I've Learned
  • Da Rant Sheet
  • Michael Fowler's Blog
  • Living in Japan with Ceoliac Disease
  • mkmaren's Blog
  • MJ
  • kcmcc's Blog
  • x1x_Stargirl_x1x's Blog
  • AuntT's Blog
  • Joe pilk
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  • My Blog
  • snash7805's Blog
  • GlutenFreeLexi's Blog
  • drewsant's Blog
  • SadAndSick's Blog
  • HONG KONG GLUTEN, WHEAT FREE PRODUCTS
  • Guth 101's Blog
  • YoAdrianne66's Blog
  • Gail Marie's Blog
  • Healthy Food Healthy You
  • SydneyT1D - Diabetic and Celiac YouTuber!
  • GFGF's Blog
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  • SMAS: www.celiac.com
  • gardener1's Blog
  • Naezer's Blog
  • JordanBattenSymons' Blog
  • JillianC
  • Sugar's Blog
  • Blanche22's Blog
  • Jason's Blog
  • Gluten-Free Sisters :)
  • Eab12's Celiac Blog
  • ohiodad's Blog
  • Newly Self Diagnosed?
  • misscorpiothing's Blog
  • anshika_0204's Blog
  • Petroguy
  • abqrock's Blog
  • WhoKnew?'s Blog
  • Soap Opera Central
  • nurcan's Blog
  • Cindy's Blog
  • Daughter_of_TheLight's Blog
  • nopastanopizza's Blog
  • w8in4dave's Blog
  • Mr J's Blog
  • Rachel Keating's Blog
  • paige_ann246's Blog
  • krisb's Blog
  • deetee's Blog
  • CAC's Blog
  • EmilyLinn7's Blog
  • Teri Kiefer's Blog
  • happyasabeewithceliac's Blog
  • quietmorning01's Blog
  • jaimekochan's Blog
  • Cheryl
  • Seosamh's Blog
  • donna mae's Blog
  • Colleen's blog
  • DawnJ's Blog
  • Gluten Challenge
  • twins2's Blog
  • just trying to feel better's Blog
  • Celiac Teen
  • MNBelle blog
  • Gabe351's Blog
  • moosemalibu's Blog
  • Coeliac Disease or Coeliac Sprue or Non Tropical Sprue
  • karalto's Blog
  • deacon11's Blog
  • Nyxie's Blog
  • Swpocket's Blog
  • threeringfilly's Blog
  • Madison Papers: Living Gluten-Free in a Gluten-Full World
  • babinsky's Blog
  • prettycat's Blog
  • Celiac Diagnosis at Age 24 months in 1939
  • Sandy R's Blog
  • mary m's Blog
  • Jkrupp's Blog
  • Oreo1964's Blog
  • keyboard
  • Louisa's Blog
  • Guts & Brains
  • Gluten Free Betty
  • Jesse'sGirl's Blog
  • NewMom's Blog
  • Connie C.'s Blog
  • garden girl's Blog
  • april anne's Blog
  • 4xmom's Blog
  • benalexander60's Blog
  • missmyrtle's Blog
  • Jersey Shore wheat no more's Blog
  • swezzan's Blog
  • aheartsj's Blog
  • MeltheBrit's Blog
  • glutenfreecosmeticcounter
  • Reasons Why Tummy tuck is considered best to remove unwanted belly fat?
  • alfgarrie's Blog
  • SmidginMama's Blog
  • lws' Blog
  • KMBC2014's Blog
  • Musings and Lessons Learned
  • txwildflower65's Blog
  • Uncertain
  • jess4736's Blog
  • deedo's Blog
  • persistent~Tami's Blog
  • Posterboy's Blog
  • jferguson
  • tiffjake's Blog
  • KCG91's Blog
  • Yolo's Herbs & Other Healing Strategies
  • scrockwell's Blog
  • Sandra45's Blog
  • Theresa Marie's Blog
  • Skylark's Blog
  • JessicaB's Blog
  • Anna'sMommy's Blog
  • Skylark's Oops
  • Jehovah witnesses
  • Celiac in Seattle's Blog
  • March On
  • honeybeez's Blog
  • The Liberated Kitchen, redux
  • onceandagain's Blog
  • JoyfulM's Blog
  • keepingmybabysafe's Blog
  • To beer, with love...
  • nana b's Blog
  • kookooto's Blog
  • SunnyJ's Blog
  • Mia'smommy's Blog
  • Amanda's Blog
  • jldurrani's Blog
  • Why choosing Medical bracelets for women online is the true possible?
  • Carriefaith's Blog
  • acook's Blog
  • REAGS' Blog
  • gfreegirl0125's Blog
  • Gluten Free Recipes - Blog
  • avlocken's Blog
  • Thiamine Thiamine Thiamine
  • wilbragirl's Blog
  • Gluten and Maize-Free (gluten-free-MF)
  • Elimination Diet Challenge
  • DJ 14150
  • mnsny's Blog
  • Linda03's Blog
  • GFinDC's Blog
  • Kim UPST NY's Blog
  • cmc's Blog
  • blog comppergastta1986
  • JesikaBeth's Blog
  • Melissa
  • G-Free's Blog
  • miloandotis' Blog
  • Confessions of a Celiac
  • Know the significance of clean engine oil
  • bobhayes1's Blog
  • Robinbird's Blog
  • skurtz's Blog
  • Olivia's Blog
  • Jazzdncr222's Blog
  • Lemonade's Blog
  • k8k's Blog
  • celiaccoach&triathlete's Blog
  • Gluten Free Goodies
  • cherbourgbakes.blogspot.com
  • snow dogs' Blog
  • Rikki Tikki's Blog
  • lthurman1979's Blog
  • Sprue that :)'s Blog
  • twinkletoes' Blog
  • Ranking the best gluten free pizzas
  • Gluten Free Product
  • Wildcat Golfer's Blog
  • Becci's Blog
  • sillyker0nian's Blog
  • txplowgirl's Blog
  • Gluten Free Bread Blog
  • babygoose78's Blog
  • G-freegal12's Blog
  • kelcat's Blog
  • Heavy duty 0verhead crane
  • beckyk's Blog
  • pchick's Blog
  • NOT-IN-2gluten's Blog
  • PeachPie's Blog
  • Johny
  • Breezy32600's Blog
  • Edgymama's Gluten Free Journey
  • Geoff
  • audra's Blog
  • mfrklr's Blog
  • 2 chicks
  • I Need Help With Bread
  • the strong one has returned!
  • sabrina_B_Celiac's Blog
  • Gluten Free Pioneer's Blog
  • Theanine.
  • The Search of Hay
  • Vanessa
  • racecar16's Blog
  • JCH13's Blog
  • b&kmom's Blog
  • Gluten Free Foodies
  • NanaRobin's Blog
  • mdrumr8030's Blog
  • Sharon LaCouture's Blog
  • Zinc, Magnesium, and Selenium
  • sao155's Blog
  • Tabasco's Blog
  • Amanda Smith
  • mmc's Blog
  • xphile1121's Blog
  • golden exch
  • kerrih's Blog
  • jleb's Blog
  • RUGR8FUL's Blog
  • Brynja's Grain Free Kitchen
  • schneides123's Blog
  • Greenville, SC Gluten-Free Blog
  • ramiaha's Blog
  • Kathy P's Blogs
  • rock on!'s Blog
  • Carri Ninja's Blog
  • jerseygirl221's Blog
  • Pkhaselton's Blog
  • Hyperceliac Blog
  • abbiekir's Blog
  • Lasister's Thoughts
  • bashalove's Blog
  • Steph1's Blog
  • Etboces
  • Rantings of Tiffany
  • GlutenWrangler's Blog
  • kalie's Blog
  • Mommy Of A Gluten Free Child
  • ready2go's Blog
  • Maureen
  • Floridian's Blog
  • Bobbie41972's Blog
  • Everyday Victories
  • Intolerance issue? Helpppp!
  • Feisty
  • In the Beginning...
  • Cheri46's Blog
  • Acne after going gluten free
  • sissSTL's Blog
  • Elizabeth19's Blog
  • LindseyR's Blog
  • sue wiesbrook's Blog
  • I'm Hungry's Blog
  • badcasper's Blog
  • M L Graham's Blog
  • Wolicki's Blog
  • katiesalmons' Blog
  • CBC and celiac
  • Kaycee's Blog
  • wheatisbad's Blog
  • beamishmom's Blog
  • Celiac Ninja's Blog
  • scarlett54's Blog
  • GloriaZ's Blog
  • Holly F's Blog
  • Jackie's Blog
  • lbradley's Blog
  • TheSandWitch's Blog
  • Ginger Sturm's Blog
  • The Struggle is Real
  • whataboutmary's Blog
  • JABBER's Blog
  • morningstar38's Blog
  • Musings of a Celiac
  • Celiacchef's Blog
  • healthygirl's Blog
  • allybaby's Blog
  • MGrinter's Blog
  • LookingforAnswers15's Blog
  • Lis
  • Alilbratty's Blog
  • 3sisters' Blog
  • MGrinter's Blog
  • Amanda
  • felise's Blog
  • rochesterlynn's Blog
  • mle_ii's Blog
  • GlamourGetaways' Blog
  • greendog's Blog
  • Tabz's Blog
  • Smiller's Blog
  • my vent
  • newby to celiac?'s Blog
  • siren's Blog
  • myraljo's Blog
  • Relieved and confused
  • carb bingeing
  • scottish's Blog
  • maggiemay832's Blog
  • Cristina Barbara
  • ~~~AnnaBelle~~~'s Blog
  • nikky's Blog
  • Suzy-Q's Blog
  • mfarrell's Blog
  • Kat-Kat's Blog
  • Kelcie's Blog
  • cyoshimit's Blog
  • pasqualeb's Blog
  • My girlfriend has celiacs and she refuses to see a doctor
  • Ki-Ki29's Blog
  • mailmanrol's Blog
  • Sal Gal
  • WildBillCODY's Blog
  • Ann Messenger
  • aprilz's Blog
  • the gluten-free guy
  • gluten-free-wifey's Blog
  • Lynda MEADOWS's Blog
  • mellajane's Blog
  • Jaded's Celiac adventures in a non-celiac world.
  • booboobelly18's Blog
  • Dope show
  • Classic Celiac Blog
  • Keishalei's Blog
  • Bada
  • Sherry's blurbs
  • addict697's Blog
  • MIchael530btr's Blog
  • Shawn C
  • antono's Blog
  • Undiagnosed
  • little_d's Blog
  • Gluten, dairy, pineapple
  • The Fat (Celiac) Lady Sings
  • Periomike
  • Sue Mc's Blog
  • BloatusMaximus' Blog
  • It's just one cookie!
  • Kimmy
  • jacobsmom44's Blog
  • mjhere's Blog
  • tlipasek's Blog
  • You're Prescribing Me WHAT!?!
  • Kimmy
  • nybbles's Blog
  • Karla T.'s Blog
  • Young and dealing with celiacs
  • Celiac.com Podcast Edition
  • LCcrisp's Blog
  • ghfphd's allergy blog
  • https://www.bendglutenfree.com/
  • Costume's and GF Life
  • mjhere69's Blog
  • dedeadge's Blog
  • CeliacChoplin
  • Ravenworks' Blog
  • ahubbard83's Blog
  • celiac<3'sme!'s Blog
  • William Parsons
  • Gluten Free Breeze (formerly Brendygirl) Blog
  • Ivanna44's Blog
  • Daily Life and Compromising
  • Vonnie Mostat
  • Aly'smom's Blog
  • ar8's Blog
  • farid's Blog
  • Sandra Lee's Blog
  • Demertitis hepaformis no Celac
  • Vonnie Mostat, R.N.
  • beetle's Blog
  • Sandra Lee's Blog
  • carlyng4's Blog
  • totalallergyman's Blog
  • Kim
  • Vhips
  • twinsmom's Blog
  • Newbyliz's Blog
  • collgwg's Blog
  • Living in the Gluten Free World
  • lisajs38's Blog
  • Mary07's Blog
  • Treg immune celsl, short chain fatty acids, gut bacteria etc.
  • questions
  • A Blog by Yvonne (Vonnie) Mostat, RN
  • ROBIN
  • covsooze's Blog
  • HeartMagic's Blog
  • electromobileplace's Blog
  • Adventures of a Gluten Free Mom
  • Fiona S
  • bluff wallace's Blog
  • sweetbroadway's Blog
  • happybingf's Blog
  • Carla
  • jaru24's Blog
  • AngelaMH's Blog
  • collgwg's Blog
  • blueangel68's Blog
  • SimplyGF Blog
  • Jim L Christie
  • Debbie65's Blog
  • Alcohol, jaundice, and celiac
  • kmh6leh's Blog
  • Gluten Free Mastery
  • james
  • danandbetty1's Blog
  • Feline's Blog
  • Linda Atkinson
  • Auntie Lur: The Blog of a Young Girl
  • KathyNapoleone's Blog
  • Gluten Free and Specialty Diet Recipes
  • Why are people ignoring Celiac Disease, and not understanding how serious it actually is?
  • miasuziegirl's Blog
  • KikiUSA's Blog
  • Amyy's Blog
  • Pete Dixon
  • abigail's Blog
  • CHA's Blog
  • Eczema or Celiac Mom?'s Blog
  • Thoughts
  • International Conference on Gastroenterology
  • Deedle's Blog
  • krackers' Blog
  • cliniclfortin's Blog
  • Mike Menkes' Blog
  • Juanita's Blog
  • BARB OTTUM
  • holman's Blog
  • It's EVERYWHERE!
  • life's Blog
  • writer ann's Blog
  • Ally7's Blog
  • Gluten Busters: Gluten-Free Product Alerts by Celiac.com
  • K Espinoza
  • klc's Blog
  • Pizza&beer's Blog
  • CDiseaseMom's Blog
  • sidinator's Blog
  • Dr Rodney Ford's Blog
  • How and where is it safe to buy cryptocurrency?
  • lucedith's Blog
  • Random Thoughts
  • Kate
  • twin#1's Blog
  • myadrienne's Blog
  • Nampa-Boise Idaho
  • Ursa Major's Blog
  • bakingbarb's Blog
  • Does Celiac Cause Sensitivites To Rx's?
  • delana6303's Blog
  • psychologygrl25's Blog
  • Alcohol and Celiac Disease
  • How do we get it???
  • cooliactic_BOOM's Blog
  • GREAT GF eating in Toronto
  • Gluten-free Food Recommendations!
  • YAY! READ THIS!!
  • BROW-FREE DIET BLOG
  • carib168's Blog
  • A Healing Kitchen
  • Shawn s
  • AZ Gal's Blog
  • mom1's Blog
  • The Beginning - The Diagnosis
  • PeweeValleyKY's Blog
  • solange's Blog
  • Cate K's Blog
  • Layered Vegetable Baked Pasta (gluten-free Vegetarian Lasagna)
  • Gluten Free Teen by Ava
  • mtdawber's Blog
  • sweeet_pea's Blog
  • DCE's Blog
  • Infertility and Celiac Disease
  • What to do in the Mekong Delta in 1 Day?
  • glutenfreenew's Blog
  • Living in the Garden of Eden
  • toddzgrrl02's Blog
  • redface's Blog
  • Gluten Free High Protein
  • Ari
  • Great Harvest Chattanooga's Blog
  • CeliBelli's Blog
  • Aboluk's Blog
  • redface's Blog
  • Being in Control of Your Gluten-Free Diet on a Cruise Ship
  • jayshunee's Blog
  • lilactorgirl's Blog
  • Yummy or Yucky Gluten-Free Foods
  • Electra's Blog
  • Cocerned husband's Blog
  • lilactorgirl's Blog
  • A Little History - My Celiac Disease Diagnosis
  • How to line my stomach
  • sewfunky's Blog
  • Oscar's Blog
  • Chey's Blog
  • The Fun of Gluten-free Breastfeeding
  • Dawnie's Blog
  • Sneaky gluten free goodness!
  • Chicago cubs shirts- A perfect way of showing love towards the baseball team!
  • Granny Garbonzo's Blog
  • GFzinks09's Blog
  • How do I get the Celiac.com podcast on my mp3 player?
  • quantumsugar's Blog
  • Littlebit's Blog
  • Kimberly's Blog
  • Dayz's Blog
  • Swimming Breadcrumbs and Other Issues
  • Helen Burdass
  • celiacsupportnancy's Blog
  • Life of an Aggie Celiac
  • kyleandjra.jacobson's Blog
  • Hey! I'm Not "Allergic" to Wheat!
  • FoOdFaNaTic's Blog
  • Wendy Cohan, RN's Gluten-Free and Dairy-Free Cooking Classes
  • Lora Derry
  • Dr. Joel Goldman's Blog
  • The Ultimate Irony
  • Lora Derry
  • ACK514's Blog
  • katinagj's Blog
  • What Goes On, Goes In (Gluten in Skin Care Products)
  • What’s new in hydraulic fittings?
  • cannona3's Blog
  • citykatmm's Blog
  • Adventures in Gluten-Free Toddling
  • tahenderson67's Blog
  • The Dinner Party Drama—Two Guidelines to Assure a Pleasant Gluten-Free Experience
  • What’s new in hydraulic fittings?
  • sparkybear's Blog
  • justbikeit77's Blog
  • To "App" or Not to "App": The Use of Gluten Free Product List Computer Applications
  • Onangwatgo
  • Raine's Blog
  • lalla's Blog
  • To die for Cookie Crumb Gluten-Free Pie Crust
  • DeeTee33's Blog
  • http://glutenfreegroove.com/blog/
  • David2055's Blog
  • Gluten-Free at the Fancy Food Show in San Francisco
  • Kup wysokiej jakości paszporty, prawa jazdy, dowody osobiste
  • Janie's Blog
  • Managing Hives & Gluten Allergies
  • Bogaert's Blog
  • Janie's Blog
  • RaeD's Blog
  • Dizzying Disclaimers!
  • Dream Catcher's Blog
  • PinkZebra's Blog
  • Hibachi Food and Hidden Gluten Hazards (How to Celebrate Gluten-Free)
  • jktenner's Blog
  • OhSoTired's Blog
  • PinkZebra's Blog
  • gluten-free Lover's Blog
  • Gluen Free Health Australia
  • Melissamb21's Blog
  • Andy C's Blog
  • halabackgirl9129's Blog
  • Liam Edwards' Blog
  • Celiac Disease in Africa?
  • Suz's Blog
  • Gluten-Free Fast Food
  • mis_chiff's Blog
  • gatakat's Blog
  • macocha's Blog
  • Newly Diagnosed Celiacs Needed for Study in Chicago
  • Poor Baby's Blog
  • the loonie celiac's Blog
  • jenlex's Blog
  • Sex Drive/Testosterone can be Depleted by Certain Foods
  • samantha79's Blog
  • 21 Months into the Gluten-free Diet
  • WashingtonLady's Blog-a-log
  • James S. Reid's Blog
  • Living with a Gluten-Free Husband
  • runner girl's Blog
  • kp3972's Blog
  • ellie_lynn's Blog
  • trayne91's Blog
  • Gluten-free Lipstick!
  • Nonna2's Blog
  • Schar Chocolate Hazelnut Bar (Gluten-Free)
  • pnltbox27's Blog
  • Live2BWell's Blog
  • melissajohnson's Blog
  • nvsmom's Blog
  • Diagnosed with Celiac Disease and Still Sick
  • snowcoveredheart's Blog
  • Gluten Free Nurse
  • Gluten-Free Frustration!
  • Melody A's Blog
  • novelgutfeeling's Blog
  • Trouble Eating Out Gluten-Free...Good or Bad?!
  • dilsmom's Blog
  • theceliachusband's Blog
  • amanda2610's Blog
  • Pancreas and Celiac Disease Link?
  • epiphany's Blog
  • Patty55's Blog
  • The Latest Gluten-Free Food Recalls
  • kenzie's blog
  • CVRupp's Blog
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  1. Celiac.com 01/25/2023 - Studies that have tried to measure the effects of a gluten-free diet on the clinical, biochemical and psychological condition of youths with both type 1 diabetes and celiac disease have delivered mixed results. A team of researchers recently set out to evaluate the impact of gluten-free diet on growth, metabolic control and quality of life in children and adolescents with type 1 diabetes and celiac disease. The research team included Enza Mozzillo, Roberto Franceschi, Francesca Di Candia, Francesco Maria Rosanio, Letizia Leonardi, Ludovica Fedi, Valentina Rosà, Vittoria Cauvin, Adriana Franzese, and M. Loredana Marcovecchio. They are variously affiliated with theDepartment of Translational Medical Science, Section of Pediatrics, Regional Center of Pediatric Diabetes, Federico II University of Naples, Naples, Italy; the Department of Pediatrics, S. Chiara Hospital in Trento, Italy; the Pediatric Diabetology Unit, Pediatric Department, S. Chiara General Hospital inTrento, Italy; and the Department of Paediatrics, University of Cambridge and Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK. The team first performed a systematic search of studies published in the last 15 years. They used PICOS framework to inform the selection process, and assessed evidence using the GRADE system. Their systematic review included only studies of moderate-high evidence quality level and reporting data on objectively assessed adherence to a gluten-free diet. Their findings highlight pre-adult adherence to a gluten-free diet in youth with type 1 diabetes and celiac disease leads to regular growth, stable BMI, without any negative effect on HbA1c and insulin requirements. Their main finding was that patients who followed a gluten-free diet experienced regular growth without any adverse increase in BMI. Moreover, the gluten-free diet does not negatively affect HbA1c and insulin, but is associated with higher post-meal glucose levels. Evidence from several studies indicate that a gluten-free diet is associated with better lipid profile and major quality of life and the psychological condition of juveniles with both type 1 diabetes and celiac disease. This study offers strong evidence that a gluten-free diet offers major benefits to juveniles with both type 1 diabetes and celiac disease. Read more at Diabetes Research and Clinical Practice.
  2. Celiac.com 01/07/2023 - First reported in 1925, the coexistence of diabetes mellitus and celiac disease is not a new phenomenon. However, as late as 1984, the connection was viewed with skepticism. Prior to the use of intestinal biopsies, some researchers reported an increase of diabetes mellitus among family members of celiac patients, while others reported contradictory findings. After development of techniques for taking jejunal biopsies, modestly increased rates of diabetes were reported. These numbers were usually close to 1% of the celiac patients who were reported as having both diseases. When faced with these increased numbers of diabetes mellitus among celiac families and celiac patients, many asserted that certain genetically determined features of the immune system (HLA) were common among people with either condition. Such genetic factors, they argued, might predispose to autoimmune diseases in general, and would therefore be likely to increase the frequency of both conditions within the same families, and sometimes, within the same individual. The issue was further confounded by the overlap between classical symptoms of celiac disease and diabetes mellitus. Thus, a second diagnosis was often overlooked because all symptoms were considered to result from the first condition diagnosed. The reported rates of coexistence thus underrepresented the true overlap. It was only through systematic research among large numbers of diabetic patients who were biopsied, along with the development and use of serological testing for celiac disease, that the increased coexistence of these diseases gained recognition during the mid to late 1980s. Still, the dominant view ascribed a common origin for both autoimmune conditions. Thus, when other autoimmune diseases were recognized as overly common among celiac patients, little thought was given to the notion that gluten might trigger more than celiac disease. Several startling research findings threatened this perspective and offered a new window through which to view celiac disease and autoimmunity in general. Although this work was initially paid little attention, each of several distinct areas of research were building toward a critical mass. Exploration of intestinal permeability, and its relationship to celiac disease, was investigated throughout the 1990s. This work not only provided convincing evidence for the use of sugar absorption tests to screen for celiac disease in developing nations, it also established that intestinal leakage of food proteins is a consistent feature of active celiac disease. One spin-off result of this work was to increase the credibility of molecular mimicry as a dynamic that followed the leakage of food proteins into the bloodstream. These foreign proteins were shown to cause immune system reactions that damaged both the foreign proteins and self tissues with similar protein structures. Meanwhile, another area of research was increasing the use of serum antibody testing, called celiac panels, which soon revealed that celiac disease was dramatically more common than previously believed. These tests also established that the coexistence of celiac disease and diabetes mellitus was also common. When diabetic patients were tested, about 5% were shown to have celiac disease. When celiac patients were tested, about 10% were found to have diabetes mellitus. Developments in similar serum antibody testing, related other autoimmune diseases, also revealed that compliance with a gluten-free diet, following diagnosis of celiac disease, actually resulted in reductions of these other antibodies. These studies suggested that gluten might actually play a causal role, not only in diabetes, but in a wide range of autoimmune diseases that were previously considered to be coincidentally associated with celiac disease. Hope dawned for many who suffered from a wide range of autoimmune diseases . Simple dietary changes might aid new treatments and bring relief to these many sufferers of autoimmunity. Even in cases where gluten is not the underlying cause, we are gaining understanding of the dynamics by which autoimmunity develops. The crowning moment, with respect to diabetes, came in January of this year when A.J. MacFarlane, et al. published their findings in the Journal of Biological Chemistry. Their work demonstrated that wheat proteins might be a major causal factor in at least some cases of diabetes mellitus. Rooted in prior animal research that was largely spearheaded by Fraser Scott, this most recent research identified immune reactions, in diabetic humans, against wheat proteins which were also closely linked with the attack on the islet cells of the pancreas. This is an exciting moment for those with type 1 diabetes. Many of them may be aided in the maintenance of islet cell transplants (another new development) through following a gluten-free diet. This work also provides a potential turning point for everyone who suffers from autoimmunity. Thanks to the recent establishment of celiac disease as very common in North America, we now know that about 1 in 133 Americans have celiac disease. We also know that this common condition is triggered by gluten. This family of grain proteins also appears to cause some, perhaps many, cases of diabetes mellitus. The evidence against gluten is also growing in the investigation of other autoimmune conditions. It appears that celiac disease will soon be the window through which we will gain a better understanding of autoimmunity, and the gluten-free diet may be the cornerstone of the treatment of many cases of autoimmune diseases.

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  4. Is there a Type I and Type II Celiac Disease IF so what is your Type: What the Diabetic Model can tell us about Celiac Disease Subtypes? ****these opinions are my own and are not an endorsement by Celiac.com….these are only my conclusions after years of research. I am sharing to generate thought, feedback, opinions, progress on the topic and change if they make sense to you. I would say there are two types of Celiac disease based on all the available research I have read. Think Type I and Type II like diabetes. I share/post this for your feedback, education, careful thought, insight and opinion(s). Maybe it will spur your thinking! This Model of a "Type I" and "Type II" Celiac disease explains best what is happening IMO! These type of grand break though ideas deserve to be shared on Celiac.com.....I suspect it will be another 10 or 15 years (IF then) that this "Working Hypothesis" will be confirmed with more studies proving IT is a "Working Theory"....I just hope some intrepid medical who might read this will be courageous enough to do a a thorough study on this topic confirming my many years of research as the Lord has been my help! I only ask that you be Berean in your thought! as you read this Posterboy blog post and Consider what I say.... Acts 17:10, 11 10 And the brethren immediately sent away Paul and Silas by night unto Berea: who coming thither went into the synagogue of the Jews. 11 These were more noble than those in Thessalonica, in that they received the word with all readiness of mind, and searched the scriptures (research) daily, whether those things were so. Type I happens as a kid (refractory often)... and Type II happens as an adult (often triggered by PPIs or Stress) see below... and why kid's don't get better (probably)....the age you get these deficiencies effects your bodies response.... triggered, in part, by congenital Vitamin deficiencies in children IMO. I concluded this based on research I recently discovered about how Riboflavin aka Vitamin B2 can affect Villi formation in children…… and why it was first diagnosed in Children in IMO. See these links on Riboflavin....I will summarize....if you get low Riboflavin, Thiamine etc. as a kid...you villi won't recover but if it happens as an adult you can recover from PPIs (low stomach acid) triggered Celiac disease. Here is the research on Riboflavin and Thiamine and Niacin and how they work together to regulate our immune system. https://pubmed.ncbi.nlm.nih.gov/7857908/ https://www.ncbi.nlm.nih.gov/pubmed/8785207 https://www.sciencedirect.com/science/article/pii/S0278691510002474 thiamine and riboflavin together https://www.sciencedirect.com/science/article/abs/pii/S1734114016302729 thiamine, riboflavin and niacin together completely down regulated inflammation/toxicity... https://www.ncbi.nlm.nih.gov/pubmed/30903555 If this Posterboy blog post doesn’t make sense to you? It will have to be someone else to rediscover this again in 15 to 20+ years... I have tried to educate the best I could! Too Educate is Too Free!!!! I have tried to share how supplementing with B-Vitamins helped my GI problems, but it seems/seemed to be a “bridge to far” for some…. May this Posterboy blog post....help you "bridge" over the connection between Genetics and Environment IE....Epigenetics...(STRESS) To discovery this I haven't cared what the truth is....I have just wanted it as it is! Truth Frees us from Error! I only know that...... Magnesium, Thiamine, Riboflavin and Niacin(amide) changed my life! ****note this comments that follows are notes to myself that quickly summarize these points (written previously) as cliff notes version of the above...(this I wrote for you as a summary for myself) "Low stomach acid triggered by stress and we lock in with antacids.. Then being low in stomach aid makes you low in Magnesium in 6 months.. Being low in Magnesium makes thiamine unavailable to the body When thiamine is low – you get low in Riboflavin after a month.. And villi begin swelling --- affecting absorption of other nutrients.. Then because riboflavin is a cofactor for Niacin synthesis via the Kyneurine pathway…. and you develop perfuse Skin Lesions diagnosed as DH… When low in meat and dairy (or take mitochondria toxic medicine) you develop pellagra when you can’t synthesize niacin from low tryptophan and riboflavin levels…" This has recently been confirmed in IMO by the discovery that Tryptophan can be used as Therapeutic in the treatment of Celiacs… https://www.news-medical.net/news/20201022/Tryptophan-found-in-turkeys-can-accelerate-intestinal-healing-in-people-with-celiac-disease.aspx Otherwise you rest/settle on just having (PSP) aka Pellagra Sine Pellagra (think thyroid problems) Or Pellagra without Skin Involvements… https://pubmed.ncbi.nlm.nih.gov/3809170/ If this is triggered by infantile Beri Beri or more probably a Riboflavin deficiency your Villi might never recover... SIDS has been linked to a thiamine deficiency in kids. Heck even in old age Beri Beri will cause blindness due to glaucoma...and 80pct of WE (brain version of Beri Beri) is not diagnosed until autopsy upon death... https://www.ncbi.nlm.nih.gov/pubmed/485004 Here is link https://academic.oup.com/ajcn/article/77/6/1352/4689829 focus on the riboflavin and gastrointestinal development paragraph... quoting from that paragraph (read it all) it very informative... "Recent work has shown that even when riboflavin is supplied to tissues intraperitoneally, the absence of riboflavin from the lumen of the gastrointestinal tract from the time of weaning leads to a disruption of normal gastrointestinal development in rats." Meaning in young children probably 2 or 3 years or young never (maybe up to 5 years of age estimate only) (fully) develop Villi if their mother was also low in Riboflavin.... cleft palates develop this way....Riboflavin deficiencies in new born baby's.....Vitamin deficiencies passed from mother to baby.... The below link was recently rediscovered by me even though it is almost 10 years people don't know its role in regulating our immune system our helping keep our GI tract healthy Here is Riboflavin’s role in protecting against the Heatshock protein in SIBO https://www.sciencedirect.com/science/article/pii/S0278691510002474 Entitled "Riboflavin protects mice against liposaccharide-induced shock through expression of heat shock protein 25" in SIBO. This also probably happening in Celiac disease as well…..we can surmise if it helps one of the GI disease’s that mimic Celiac disease….it could help Celiac disease as well. See this old forgotten article about how SIBO is common in Celiac disease. Entitled Bacterial Overgrowth of Small Intestine aka SIBO Common in Treated Celiac Disease https://www.celiac.com/celiac-disease/bacterial-overgrowth-of-small-intestine-common-in-treated-celiac-disease-r791/ Note: a couple things here….this Is not suspected Celiac disease but “Treated Celiac Disease”….. SIBO is common in treated Celiac disease….meaning these were people eating a gluten free diet already…. And as I often site and say in my Posterboy blog post this research 10 or 15+ years old…. And yet people (doctor’s/clinician’s) are not aware of these connections/associations…. I set out to confirm my diagnosis and it took me approx. 4+ years to realize I had Low/NO stomach acid going un/misdiagnosed. I have continued studying since then….and I am convinced now more than ever 10 to 12+ years later that Low/NO stomach acid could help Celiac’s….. But we have a “Long Tail” Memory…..and can’t seem to move onto another possible diagnosis like Low/NO stomach acid despite what the research seems to say! I am not the first to discover, study or conclude this… This was studied 30+ years ago....but through much study and the grace of God I have rediscovered it..... will it be another 10 or 15 years before it is accepted??? I hope and pray not! But I honestly don't have much hope.... That people will read it and believe it.....it has been lost for 30+ years for a reason.....nobody believes it! See this research entitled Gastric morphology and function in dermatitis herpetiformis and in C(o)eliac disease. https://pubmed.ncbi.nlm.nih.gov/3992169/ quoting “Antrum-sparing chronic atrophic gastritis was present in 92% of the achlorhydric patients, and hypergastrinaemia and serum parietal cell antibodies were found in most of them. The prevalence of chronic gastritis of the body and of the antrum increased with age.” Note again: this was not in NCGS patients….but diagnosed Celiac and DH patients…. Surely Low/NO stomach acid is the trigger for Celiac disease. IF this is so we would see the same thing in PPIs users… And WE do….recent research confirmed this analysis….among PPIs users….for those who used them for a year or more Low Stomach Acid (from PPIs Acid reducers) were found to be the trigger (CAUSE) of their Celiac diagnosis! See this article about it on Celiac.com entitled “Do Proton Pump Inhibitors Increase Risk of Celiac Disease” https://www.celiac.com/celiac-disease/do-proton-pump-inhibitors-increase-risk-of-celiac-disease-r2860/ They summarize it very well…. Quoting “The data clearly show that patients who use anti-secretory medications are at much greater risk for developing celiac disease following the use of these medicines. The fact that this connection persisted even after the team excluded prescriptions for anti-secretory medicines in the year preceding the celiac disease diagnosis suggests a causal relationship.” See also this thread started by Knitty Kitty https://www.celiac.com/forums/topic/117685-atrophy-associated-with-ppis-nsaids-and-ssris/ Back to the main point of this Posterboy blog post….Low stomach is triggerable and reversible a “Type II” Celiac disease. A congenital Celiac disease diagnosed as a kid…could not be reversed (in most people)…. and why it was originally diagnosed as a “Type I” genetic disease once Celiac antibodies were discovered IMHO. I don’t believe it fair to someone who at 40 or 50 started taking PPIs to then tell them…you now have a genetic disease…. No, it was triggered by your PPIs usage….and is reversible like most EPI (Environmentally) genetic triggered diseases are…. B-Vitamins are the very definition of Epigenetics….you got low in Vitamins….and you got a disease from the deficiency…. This might not show up for years….until the Doctor’s recognize as a deficiency…..hence a “Type II” diagnosis! I have written a couple other Posterboy blog posts that might help you. An “Open Letter” Part 1 and Part 2 that explains how these things are connected if you think it would help you to read some more on these connections. I didn’t understand it at the beginning of my journey, but I hope it will help start you on your journey back to heath as it did me! It has been a long journey for me….but It can be short(er) for you….because you know the way back….I have shown you how I got back! https://www.celiac.com/blogs/entry/2167-open-letter-part-1-to-fellow-gi-sufferers-etc-like-ibs-uc-and-other-gi-diseases-like-infant-heartburn-gerd-that-grows-into-in-time-to-ibs-uc-chrons-and-ncgs-as-a-teenager-or-celiac-disease-in-time-as-an-adult-look-beyond-to-the-parent-disease/ https://www.celiac.com/blogs/entry/2515-open-letter-to-the-many-gi-sufferers-part-2-still-suffering-look-beyond-these-symptom%E2%80%99s-to-the-parent-disease-pellagra-with-these-many-unruly-children-like-ibs-gerd-uc-etc-up-to-and-including-ncgs-and-celiac-disease-in-time-i-believe-part-2/ Just trying to help those still suffering (I believe) unnecessarily. Who think there is only one Type of Celiac disease? 2 Corinthians (KJV) 1:3,4 3) “Blessed be God, even the Father of our Lord Jesus Christ, the Father of mercies, and the God of all comfort; 4) who comforteth us in all our tribulation, that we may be able to comfort them which are in any trouble, by the comfort wherewith we ourselves are comforted of God.” I hope this is helpful but it is not medical advice. 2 Timothy 2:7 As always, “Consider what I say; and the Lord give thee understanding in all things” this included. Posterboy by the Grace of God, ADDENDUM: I am adding this Posterboy blog post from an earlier blog post maybe it will help you…. https://www.celiac.com/blogs/entry/2103-why-and-how-pellagra-is-often-confused-with-celiac-disease-andor-other-gidigestive-problems-the-science-of-pellagra-a-hidden-epidemic-in-the-21st-century-presentingrevealing-as-ncgs-andor-possbily-celiac-disease/ At the time not many people read it…..but maybe this time more people will read and understand it! The presenting symptom of DH in Celiac disease is confirmation that the Kynurenine Pathway has broken down and who have developed the 3rd "D" of Pellagra…. Let’s hope the doctors realize this before the 4th "D" symptom DEATH occurs! Since 80 percent of Wernicke Encephalopathy better known as the mental/brain symptom of Beri Beri is diagnosed on autopsy I have my doubts they will notice the Pellagra Co-morbid in 58% (Majority) of Celiac noted no less than by the IJCD (see Posterboy blog post link above) and why I continued to emphasis this in my Posterboy blog posts….. because the IJCD noted the same thing! I only ask that you believe you own research (prophets)…..and agree with them that Beri Beri, Pellagra Sine Pellagra, and Pellagra is happening in Celiac’s. https://www.celiac.com/celiac-disease/how-low-thiamine-can-thin-villi-old-research-rediscovered-and-its-clinical-significance-in-celiac-disease-r5100/:~:text=Beriberi%20is%20is%20caused%20by%20low%20thiamine%20(B1).,dietary%20thiamin%20deficiency%20on%20intestinal%20functions%20in%20rats.” At what rate only more tests/studies can prove. I ask that others in the Celiac community that might read this…. Do these studies….so that future doctors are aware of these connections/associations. I just know we can't wait another 10 or 15 years before this trickles down to the clinical (Medical) level when current (today's) research shows that Tryptophan can be used as a Therapeutic for Celiac disease.....IMHO confirming this theory! https://www.celiac.com/celiac-disease/tryptophan-in-turkey-meat-can-speed-gut-healing-in-celiac-disease-r5338/ Isn't that we ALL want to speed our healing! This is happening in other auto-immune diseases too/also because we only have ONE immune system! With many difference (faces)….IE symptom’s….. Note: Pellagra is found in Lupus too! https://www.celiac.com/blogs/entry/2709-the-lupus-inflammation-model-for-sickness-beginning-with-b-vitamin-deficiencies-in-celiacs-and-other-inflammation-triggered-diseases%E2%80%A6/ I must stop for now, or this will be way too long again!
  5. Celiac.com 04/11/2022 - Researchers and clinicians are just beginning to understand the many connections between celiac disease and diabetes. We've done a number of articles on links between celiac disease and diabetes. We've talked about how a gluten-free diet might help lower diabetes risk. We've looked at the potential role of gluten in Type 1 Diabetes. We've even asked if having type 1 diabetes mellitus and celiac disease automatically mean worse health and quality of life? We know that rates of diabetes are much higher in celiacs than in the general population, and vice versa. We also know that non-diabetic patients often show celiac-specific humoral immunoreactivity at the time of their celiac disease diagnosis. What about diabetic patients? Do they show type 1 diabetes celiac-specific humoral immunoreactivity? To find out, a research team recently looked at celiac-associated humoral autoimmunity in child, adolescent, and adult patients at the onset of type 1 diabetes (DM1) to see if those patients exhibit DM1 celiac-specific humoral immunoreactivity, as do non-diabetic celiac patients at first diagnosis. The research team included Claudio Tiberti Aceliac disease, Francesca Panimolle BS, Margherita Bonamico MD, Blegina Shashaj MD, Tiziana Filardi MD, Federica Lucantoni BS, Raffaella Nenna MD, Francesco Costantino MD, Andrea Lenzi MD, and Susanna Morano MD. They are variously affiliated with the Department of Internal Medicine, University of Rome “Sapienza,” Rome, Italy; the Department of Pediatrics, University of Rome “Sapienza,” Rome, Italy; and the Department of Physiopathology, University of Rome “Sapienza,” Rome, Italy. The team found IgA anti-transglutaminase autoantibodies (IgA-tTGAb) in more than 650 new-onset DM1 serum samples. They then analyzed IgA-tTGAb-positive DM1 samples for IgG-tTG, deamidated gliadin (DGP), and actin antibodies, and compared the results against those from more than 80 screen-detected non-diabetic patients at the time of their celiac diagnosis. In all, nearly thirteen percent of DM1 samples were positive for IgA-tTGAb, with patients 18 years or over showing lower autoantibody frequency, that's about 2.2 times more than in adult patients. Meanwhile, compared with non-diabetic celiacs, IgA-tTGAb+ DM1 patients showed substantially lower IgA-tTGAb titers, IgG-tTGAb, and DGPAb frequency/titers, along with sharply lower average number of celiac-autoantibody positive results per patient. These results show that the age of diabetes onset is negatively associated with risk of celiac disease, that is, the lower age of DM1 onset, the higher the risk of developing celiac disease. Compared with the activity of non-diabetic patients at the time of celiac diagnosis, celiac-specific humoral immunoreactivity is sharply lower at the onset of DM1. The team suggests that a general lower celiac-specific humoral immune response reflects a slower process of celiac disease development in DM1 patients, which is marked by nearly imperceptible gastrointestinal symptoms. This hypothesis is supported by an autoimmune diabetes study that shows a direct correlation between intensity of the humoral immune response and more prominent characteristics of insulin deficiency.* Stay tuned for more stories on connections between celiac disease and diabetes. Read more in Diabetes Care. 2012 Oct; 35(10): 2083–2085 *Buzzetti R, Di Pietro S, Giaccari A, et al. Non Insulin Requiring Autoimmune Diabetes Study Group High titer of autoantibodies to GAD identifies a specific phenotype of adult-onset autoimmune diabetes. Diabetes Care 2007;30:932–938
  6. Celiac.com 10/22/2021 - The iceberg starts by scratching the hull. The captain and crew had early warning signs but it all happened so quickly from there. The destruction continues on and the hull is breached, allowing water to rush in. We are seeing this happen now. People are starting to perish aren‘t they? Next, the boat starts to list to starboard. Passengers are abandoning ship while the crew goes into emergency status. The pumps are started and priorities set. It's a bit chaotic but many are still being hopeful and thinking positively. Those with vision imagine the worst-case scenario and act accordingly. "I think we can patch this thing up if we back away now and try not to drive this rock any deeper." Unfortunately, this is man's ‘M.O.", isn't it? We do have to hit the iceberg before we learn many of the important things in life. Well, we have hit them and hit them hard. As I stated so melodramatically in the opening, you are alive to see the paradigm shift that has resulted from hitting this iceberg. The ship is going down. One drug after another has been placed under "the microscope" and failed the acid test. The cans are all falling off the shelf. For those in medicine, the colon contents have hit the fan. Pick a metaphor and run with it. Thank God there are lifeboats! And there are lots of them—enough to rescue the entire compliment of passengers and crew. It's just that they are small, spread out, and there doesn't appear to be enough of them. But there are adequate provisions if we stay calm, work together, pool our resources, and WANT to survive. We have to want to get better and we have to believe that there are lifeboats that can get us to safety. The lifeboats are people who understand nutrition and how the body works. They are wise doctors who listen to their patients before prescribing medication, whether they are conventional or holistic preparations. I look at the Internet as a set of lifeboats, although some of them have holes in them. What's the expression? Oh yeah "Knowledge is power." That is partly right, for sure. Faith is extremely important, as is courage. You must believe and act upon what you think is true. You must be confident, strong, and persistent. So, we need to cover a few more things to build up your faith in the idea that we are in charge of our health destiny more than we have ever been led to believe. It's not hard to imagine now that you know about the extremely common malabsorption syndrome being induced by the staples of our diet, right? There is more, though—much more. The two biggest killers of human beings are atherosclerosis and cancer. A person every 3.5 seconds dies of a stroke or heart attack in this country. And yet, the dog does not suffer from this as a lethal, clinical entity—yet! Veterinary pathologists are seeing atherosclerotic changes in the vessels of dogs on necropsy. And this is very important for us to understand. Dogs can have atherosclerosis but they don't have it badly enough to cause clinical disease. They develop so many of the conditions that take human life, including a higher incidence of cancer, but dogs don't suffer from this deadly disease process. Why is that? Simply put, they don't get "enough" hydrogenated oils in their diet. I believe down to my socks that trans fats are the single-most important factor in the development of atherosclerosis—the "solvents" that allow things into the walls of the arteries of those that consume them and set the stage for the inflammatory process that follows. Dogs do get some trans fats in their diet, as they are fed French fries, snack foods and bits of human desserts that are loaded with these culprits. But, they clearly do not get enough to lead to a clinical syndrome. (Please read Hydrogenated Oils-The Silent Killers, by David Dewey on the Internet. Whoa! You will clearly see how and why the first recorded myocardial infarction took place only ten years after hydrogenated oils hit the shelves in the form of margarine. You will also see how and why another plague—type 2 diabetes—"adult-onset" diabetes—followed ten years after that. Hey, dogs don‘t get that one either—yet! "20:20".) But cancer??? Oh yeah, dogs have lots and lots of cancer. Why is that? Because they have plenty of the viruses that cause cancer and experience the same immune suppression as humans stemming from their diet, the environment, and the drugs they receive. "Back up for minute. That's the second time you've said viruses cause cancer." Yes, once again, there are medical researchers who have believed for over thirty years that all cancer is viral in origin—not just some, not even most, but all. I personally believe that this is true, especially after spending as much time studying about these guys as I have. And this belief positions all other "carcinogens" where I think they belong: as secondary factors, facilitating the action of the virus by causing immune suppression, chronic tissue damage, and/or damage to our actual DNA, where I believe many of these culprits reside. The "four horsemen" fit right into the clinical picture here by inducing at least two of the three factors- causing immune suppression and inciting chronic tissue inflammation and damage. The immunosuppressive effects of the big four should be easily imagined. Back to the coral reef covered with oil. How can those villi that are coated with the problem glycoproteins manage to absorb optimal levels of B complex, vitamin C, and other nutrients critical to the health of the immune system? How can they do it when they are leveled by the immune response to the glue foods in true celiac disease or the related conditions of true casein, soy, or corn intolerance? This should be a no-brainer—and it is—literally. The brain suffers tremendously from the lack of these nutrients along with a concurrent deficiency in calcium, zinc, iron, iodine, and the pandemic omega three fatty acid deficiencies that exist in humans and pets. Therefore, it shouldn't be a mystery that cancer is rampant among the three species. The fact that viruses cause cancer is a done deal in veterinary medicine and has been for quite some time. I won't bore you with the list of examples. And yet, it was not until March of this year—2005—that the American Cancer Society put viruses on their list of carcinogens. How can that be? Are you as baffled by that as I am? Remember those landers on Mars? Wherever you have immune suppression and chronic inflammation come together then you should look for cancer. Estrogens fit the bill here and therefore should not surprise anyone as leading carcinogens in breast (mammary) cancer. The good news is that the ovaries of the female do not make enough estrogen by themselves to create this dilemma. It is what the individual is consuming (including hormone replacement therapy) or exposed to in the environment (pesticides, environmental toxins, food animal additives, etc) that tips the scales in favor of the cancer (or endometriosis, polycystic ovaries, PMS, catamenial seizures, or other estrogen-related disorders). The "big four" foods fit right in here because the gluten grains, dairy, and soy are loaded with estrogens. Dairy is a huge factor here as it also provides cholesterol precursors to the formation of these hormones. Seeing a pattern here? Yes, the foods that are bad for us are so in a number of ways. So, we should not be surprised to see that Asians have a 15 times lower rate of breast cancer and a 5 times lower rate of prostate cancer—on their native diets. It is NOT because they eat soy. The truth is that they eat very little soy. But they do NOT eat dairy, wheat, or corn in their traditional diets. Just go to any authentic Japanese or Chinese restaurant and look for cheese, bread, or corn chips. Which brings us to "lectins", something I mentioned a while back. Lectin is the term that has been given to the antibody-sized glycoprotein that is derived from the consumption of foods that are part carbohydrate and part protein (thus the term glyco-protein). Once again, the big four foods are glycoproteins by structure. Our antibodies are also glycoproteins, a protein core with a sticky carbohydrate outer covering to facilitate adherence to foreign proteins such a viruses, bacteria, and the like. In fact, viruses have glycoprotein receptors on them. Normally, our antibodies attach to these sites. Hmmm—I wonder if dietary lectins ever do? Could one plausible explanation for food-induced immune-mediated disease episodes be that the chronic latent viruses in situ in our tissue become coated with dietary glycoproteins rather than our antibodies and that when we develop IgE, IgG, and other antibodies to these foods that our immune system starts to react to these "food-coated" viruses in the host tissue and attack that tissue just as it would if it were a viral infection coated with our own antibodies? Could that be how food lectins such as those from wheat, dairy, soy, or corn auto-agglutinate red blood cells. Maybe it is just the glycoprotein itself that does it in most cases but it sure would help to explain why some "autoimmune diseases" are triggered by foods while others follow viral infections, either naturally acquired or through vaccination. It would also help to explain why avoidance of the trouble foods could greatly reduce the incidence or recurrence of these attacks. (For a well-written discussion on lectins, please look up The Lectin Report on the Internet. It goes into great detail about how these tiny glycoproteins "unlock" the cell and allow things to enter it, inciting inflammation and causing cell death. It's all about the same guys. The four horsemen ride again.) The fantastic news is that sooooo much starts going right once the big four are avoided completely and for a long enough time. By avoiding the casein, gluten, soy, and corn, the gut starts to heal and the malabsorption syndrome begins to reverse. How long does it take for the intestine to heal once the offending foods are withdrawn? Well, according to the celiac literature, it takes anywhere from 6 months to 2 years for the duodenum to return to normal. Does that make sense? Not to me, unless you consider the fact that gluten is not the only thing doing the harm to those duodenal villi. This was my first quest, to get on celiac forums and make sure they knew the truth about casein, soy, and corn. I hated reading about celiacs that had struggled so valiantly to be gluten-free only to find that they were shooting themselves in the foot big time by the continued consumption of the other three culprits. Statistically, celiacs have a 50% chance of also being casein intolerant. I have to believe that it is much more common than that. But soy and corn are looming larger and larger as we fall for the myth that soy is a health food, we turn to vegetarianism for various reasons, and we continue to genetically modify corn to death. Assuming that we do enough right, the gut does heal and probably much more quickly than we currently believe. After all, it is one of the fastest healing tissues in the body. Once healed, it starts to take in all of the calcium, iron, iodine, B complex, vitamin C, and trace minerals that it has been starving for over the past years, often from the moment the individual started consuming the big four. The thyroid becomes healthy, the iron deficiency resolves, enzyme systems start operating at peak efficiency, tissue repairs, and the immune system gets back to normal. And that last item is critical. That's when many of the long-term symptoms finally resolve—the allergies, GI signs, skin problems, and in the best case scenario, the immune-mediated diseases. I would love to think that the risk of cancer then plummets, as well. What couldn't our immune system accomplish if it were in optimal condition? I can no longer put limitations of what our body is capable of doing in the way of healing or prevention when I think about that last statement. However, I know that our environment—with its staggering levels of serious pollution—is a HUGE limiting factor. I would love to dive into that topic but time constraints prevent that. The really cool thing is that some "completely unexpected" things can happen when individuals go GFCFSFCF (gluten-free, casein-free, soy-free, corn-free). In retrospect, they were "unexpected" only because we had not fully grasped the impact of what we had learned. The most notable—the most amazing—the coolest of the cool—the "hook" as I now call it—was the response of epileptics to this elimination diet. I find it just so utterly fascinating that something we stumble upon can wind up being the glaring example of everything we know—AND end up leading us into realms that we could only dream about in the past. Epilepsy is just that condition. Man, I could talk about epilepsy for the full two hours- how it all comes about and what the study of this condition has done to my knowledge base. Thankfully, I have chronicled the entire journey on my Website (www.dogtorj.com), starting with how I read the captivating fact that celiac children with epilepsy who went gluten-free often had major reductions in—if not total cessation of—their seizures. "Wow. I wonder why that happens?" I asked myself. "Epilepsy is considered idiopathic in veterinary medicine. There has to be something about wheat that leads to seizures." Elementary thinking, I know. But, this was novel stuff to me. How about you? It did not take long at all to find that MSG (monosodium glutamate) could trigger seizures and that wheat gluten was an incredible 25% glutamic acid by weight. "Eureka! Is it that simple? I then found that soy had even more glutamate, almost twice as much as wheat. "Oh, oh." Casein is 20% glutamic acid by structure. Yep, three of the four horsemen are packing glutamate in their saddlebags. Do the food sources of these neurostimulating—potentially neurotoxic—non-essential amino acids (glutamate and aspartate), really do the same thing to our brain that the "crack cocaine" versions (MSG and aspartame, respectively) do?" I assumed so and started putting my epileptic canine patients on gluten-free diets and, son-of-a-gun, they became vastly improved. Some stopped having seizures completely within 24 hours of the diet change and never seized again. We were onto something—and it was big—really big. As the significance of these findings sunk in, I threw myself into the study of neurological, psychiatric, and other "idiopathic" neurodegenerative conditions that affect us all. The "excitotoxins" ( MSG and aspartame), as Dr. Russell Blaylock termed them, were well-known culprits and played roles in epilepsy, ADHD, bipolar disease, and more. It wasn't until later that I would finally start reading about their involvement in the other "big 4": MS, ALS, Alzheimer's and Parkinson's. But still, no one was talking about the FOOD sources of these non-essential amino acids. But it was so simple, and a little something called "insomnia" illustrated the point I was trying to make. These neuroactive amino acids were clearly responsible for waking people up like a shot at 1-2 AM, 5-6 hours after eating dinner and dessert and I was a living example. And that was the exact time interval I was finding between meals and seizures in my un-medicated dogs. Once I published my work on the Web and contacted over 500 breeders in the process (oh, how I love the passion of breeders), the testimonies to these finding were flowing in on a regular basis. And, the pieces to this puzzle started fitting together—phenomenally well. And here is where it all comes together. I had written a totally different summary for the end of this discussion, but upon proofreading the pages, I realized that the discussion of epilepsy—the condition that grabbed my attention and pulled me into this epic battle at Helm's Deep—would serve that purpose. It would illustrate all of the principles that I "preach" every day in the exam room and will attempt to enlighten people with (hopefully not bore to death) in lectures like this for the rest of my days. Principle number one: The foods that are bad for us are bad in numerous ways. The "four horsemen"—gluten (from the grains wheat, barley, rye), casein, soy, and corn terrorize us in more ways than simply inducing villous atrophy, which results in the chronic malabsorption of the essential nutrients that we have covered. These foods provide staggering levels of glutamate (and aspartate), estrogens, allergens, and lectins, and when prepared for consumption, act as carriers of many of man's worst creations in the form of GMO's, hormones, and chemical additives. So these foods damage our gut, cause malnutrition of our entire body, and provide many of the ingredients necessary to generate symptoms including pain, sleeplessness, high blood pressure, behavioral disturbances, and seizures. Applying this to epilepsy, the brain suffers from the malnutrition, the immune system going down, and the rise of chronic latent viruses. The war begins. Add the vaccination with modified live virus vaccines made with viruses that love the central nervous system. These viruses naturally take up residence in the glial cells of the brain, those cells that control the level of the normal neurotransmitter—our friend glutamate—at the synapse. We have seen vaccine-induced disease in the past, right? How hard is it to believe that this is happening "sub-clinically"? These top allergy-producing foods are also stimulating histamine production, the release of which causes the blood brain barrier to become more permeable to glutamate, a normal occurrence that serves to counteract the depressing effects of histamine. These same foods contain estrogens, both naturally occurring and those from pesticide residues, which are neurostimulating, irritating, and immunosuppressive. P.M.S. anyone? How about catamenial seizures? I hear about them all of the time. The malnutrition that we have discussed then starts to compromise enzyme systems in the liver, kidneys, and elsewhere in the body, some of which are responsible for controlling the blood levels of the evil twins, glutamate and aspartate. No wonder some of us have seizures. The way I see things now, it's a bigger wonder that more of us don't have epilepsy. Are you seeing "Pandora's Box" opening yet? Have you grasped what these foods- the damage they do and the ingredients they contain- are capable of? Thank Goodness our body knows what to do with all of this mess we put it through, eh? And it does. Therefore, principle number two: Our body never makes mistakes—ever. Only we make bad choices about what we do to this vessel of ours. "What about birth defects?" is always the first challenge. Certainly, this degenerative process sometimes begins prenatally, leading to premature births and birth defects, but we are ultimately responsible for these occurrences. The more you learn about this topic (and study celiac disease as a model), the less you will simply write off to "genetics" or Providence. Fevers, heartburn, sore throats, nasal congestion, bronchoconstriction, diarrhea, hives, headaches, and even "autoimmune" attacks have a purpose. Some clinical signs are warning signs that we have made a mistake while others are therapeutic measures on our body's part. Others are both. "But autoimmune disorders?" you might question. Yes, I believe down to my socks that viruses are vitally involved in most (if not all) immune-mediated diseases. I believe that the immune-mediated diseases are our immune system's valiant attempt to wipe out these viruses before they have the opportunity to do what they really "want" to do—cause cancer. This would help to explain why people with chronic active hepatitis have such a high incidence of liver cancer. But it might also help to explain why the same breeds of dogs that develop panosteitis—the ones with all of the allergies and other juvenile bone diseases—are the guys that go on to develop bone cancer at age six and a half. So, is eosinophilic panosteitis our body's attempt to rid the bone of viruses that might later go on to cause cancer? What are eosinophils involved in other than allergic reactions? "But get back to seizures. I can't wait to hear how a seizure is a good thing", the skeptics are saying. Yes, I am convinced that even seizures serve a vital purpose; that being to burn up the excessive glutamate in the brain. As you may know, no matter what the cause of our bodily death may be, the brain dies as a result of the "glutamate cascade"—the sudden rise of glutamate in the brain resulting from the dying glial cells and increased permeability of the blood brain barrier. Glutamate is potentially—and eventually—neurolethal. How hard is it to believe that seizures are designed to keep the death of vital neurons from happening? The sufferers of ALS (Lou Gehrig's Disease) sure wish a peripheral neuron could have a seizure. But because they can't, the neurons eventually die as a result of the excess glutamate in the synapse. But guess what. ALS sufferers have also reported benefits from what I now call "The G.A.R.D"—.the glutamate-aspartate restricted diet. So have people with ADHD, chronic pain, insomnia, MS, and other conditions that have the "excitotoxins" as part of their pathophysiology. This is all on my site. Seizures may even serve to limit viral infections, if through no other mode of action other than to encourage a rise in body temperature, something that viruses hate. We know that viral infections of the central nervous system are usually accompanied by high fever, right? Remember: That's a good thing. (I wonder how many people who died of West Nile Virus might have survived if we didn't treat them so aggressively? Its a parallel to that cancer thing we talked about earlier.) So, do you see why I got so excited about the role epilepsy would play in bringing people into the fold? It has all of the elements we have discussed—all of the necessary cast, plot, and scenery to make a great and epic tale of how the four horsemen rode into town and stole our health. But we really did it to ourselves, didn't we? We made these bad choices. The fact is man created the wheat we now eat in about 400 AD, introducing lethal quantities of gluten into our diet. A millennium later, we changed milk sources from goats to cows, adding casein to the mix. Now, five hundred years later, we want to start eating soy—"the third plague"—something that has been previously relegated to the lowly positions of a nitrogen-fixing, rotational crop and a mere condiment on the table of our Oriental restaurants. Did we really just get smart enough to see the health benefits of consuming the soybeans themselves? Do we really think that loading our bodies up with plant estrogens, goitrogens, anti-nutrients, villous atrophy inducing "glues", and staggering levels of the non-essential, epileptogenic amino acid glutamate is going to improve our health. How many trips to Mars are we gonna take, anyway? We do reap what we sow. Through the eyes of food intolerance, medicine becomes so simple that even* the layperson can understand it. (* I say "even" because I know a growing number of lay people who understand these things much more than the doctors who look down their noses at the "untrained".) Isn't that the way it should be? Shouldn't we all be able to comprehend our medical lives? It is, after all, one of the most important aspects of our existence, isn‘t it? The way I look at it now, our medical lives are divided into three phases: the acquisition of viruses, the progressive malfunction of our body and immune system, and the failure of our immune system. I think you now have a very good idea why that occurs - In a word, malnutrition. Symptomatically, it usually also breaks down into three phases: allergies, immune-mediated diseases, and cancer. We see this so clearly in certain breeds of dogs but I have also heard this sort of history from many, many people I have interviewed about their own health. Hopefully how and why these three phases occur is much clearer now. The allergies—phase one.. are the warning signs that you are making mistakes. The immune system is throwing warning signs at us while closing the doors to further invasion. Phase two—the immune-mediated diseases—is the first set of conditions that these allergies were warning you of. They are also a second set of warning signs of increased severity because we didn't act upon the first set. We usually still have time to get things right owing to the fact that our organs can take a beating and still survive and repair—usually we still have time. Unfortunately, some are lost in phase two due to overwhelming lupus, glomerulonephritis, or the chronic active hepatitis that finally raised its ugly head. As I have mentioned, the last condition in that list is a glaring example of the type of condition that has led me to make some bold statements concerning the role of viruses in immune-mediated diseases AND why phase three—immune failure—often manifests as cancer. I spoke of bone cancer occurring in problem breeds at 6.5 years of age. What else happens at six to seven years of age in the dog? Better put, what doesn't happen at that age: tons of immune-mediated diseases, cruciate ruptures, spinal disorders, heart murmurs, worsening allergies, numerous benign skin tumors, and more. It's a crisis period, isn't it? Just like 40-50 years of age is in the person. If these conditions are all "genetic", why do they wait so long to show up? Hmmm—great question. Something is waiting, right? What??? Can we think of anything that we have in our bodies that might be "waiting"? I can. They're called viruses. We have been acquiring them our entire lives. Our parents even gave some to us. "What?" Can't viruses be transmitted vertically? How about genetically? Others we acquired "naturally" during our lifetime and still others we acquired through vaccination. (Most of our pets and us are too unhealthy to take on any more modified live vaccines, aren't we?) And as I have mentioned , we invited many in to stay by killing the fever that was designed to limit the infection. We have become walking virus hotels—"mobile homes" for these guys, if you will. The startling fact is that we are riddled with ‘em. And they are waiting for their chance. They are the ultimate opportunist—the consummate terrorist. Sure, there are others: bacteria, mycoplasms, fungi, and more. But the virus is the guy who incorporates his genetic material into our cells and then bosses them around. He's the guy who our immune system hates enough to risk killing our own tissue to root him out. He's the guy that can go anywhere in our body and do anything he wants ONCE we get to that point of immune suppression that we are destined to reach once we have done enough wrong to this body of ours. In my mind, he wasn't designed to be. Viruses are ubiquitous in nature and critical to its development, variety, and adaptation. So why did they turn on us? "Shoot—look at the time. We'll have to go down that rabbit hole after this presentation." I think you can figure it out, anyway. Yes, we DO have our health destiny more in our own hands than we ever believed. Yes, we DO reap what we sow. We just didn't realize that we were sowing such bad seed all of these years did we? We have had glimpses of our wrongdoing and our conscience has told us not to overindulge and to try to eat properly. That's just common sense, right? But whodathunk that the staples of our diet were killing us? Who would believe that cow's milk, wheat, and the "newest health food"—soy (errrrrh)—were plagues on mankind, brought on by our own doing? And who would believe that the "simple" elimination of the big four would lead to the vast improvements in our health that I have personally experienced. (I cannot overstate the phenomenal changes that have taken place in my body over the past 5 years) I'll tell you who would believe such things: those that know that our body does not make mistakes—ever. (Only we make mistakes in our choices of what to put into our bodies.); those that can still remember why our body does what it does instead of just covering up the symptoms should believe this (Who would believe that heartburn might be a symptom that we ate something wrong? Wow!); and those that have eyes to see and ears to hear and can still be taught something. They are fewer in number than I ever thought existed. BUT, there have been enough wise people over the years to carry this torch. There have been a select number of doctors, researchers, and lay people that have been beating this drum for years and years and stood their ground against the onslaught of drugs and misinformation—the "magic" (pharmakeia) and slight of hand—that has taken away the motivation of the masses to find real answers to their health problems. Is the white tiger really gone or does he lurk off stage, sometimes even attacking his master? Yes, celiac researchers, holistic health advocates, naturopaths, and the like have finally been vindicated. They rode out to meet the enemy years ago and are finally being joined by an ever-growing cavalry. Against seemingly insurmountable odds, the message has survived—"We are what we eat. You do have control of your health's destiny." It is through the valiant efforts of this Brotherhood—and the prevailing nature of Truth—that we have won at Helm's Deep. Now, it's on to the final battle. Go to Part 1: Food Intolerance—Man and Animals versus Gluten, Casein, Soy, and Corn or How We Won the Battle of “Helm’s Deep” (Part 1 of 2)

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  8. Celiac.com 02/08/2022 - The numerous connections between celiac disease and diabetes have led many clinicians to screen diabetes patients for celiac disease, and vice versa. The case of a five year-old girl's recent celiac diagnosis during a diabetes study further highlights that connection. Shane and Staci Vogel, an Iowa couple with a family history of diabetes, enrolled their daughter Kemper, 5, and son Knox, 2, in Sanford Health's PLEDGE study, a large-scale screening of children under age 6 for type 1 diabetes and celiac disease. Fortunately, the study found no sign of diabetes in either child, but the process did lead to a celiac diagnosis for Kemper. Kemper didn't have any clear symptoms of celiac disease at the time of her diagnosis, the family quickly moved to rid their home of gluten ingredients, and get her on a gluten-free diet. Begun a year ago, with the modest goal of using a few clinics to test 1,000 children for diabetes and celiac disease, the PLEDGE project screened more than 2,000 children in its first year, study researcher Dr. Kurt Griffin told reporters. The PLEDGE study has grown from just a few clinics to over forty-two, Griffin said, including all of Sanford Health clinics in Sioux Falls, among others. According to Sanford's website, the study is now enrolling children under 6-years old, provided they are currently receiving annual checkups at Sanford Health, and are not diagnosed with type 1 diabetes. Sanford is offering the study at no cost to families. To learn more about the PLEDGE study and whether your child qualifies, call (877) 878-4825. Read more in the Sioux Falls Argus Leader
  9. Celiac.com 11/09/2021 - A team of investigators recently set out to determine if patients with type 1 diabetes mellitus (T1DM) are at an increased risk of developing celiac disease compared with the general population. Their study, “Threshold for Undergoing Celiac Disease Diagnosis in Pediatric Type 1 Diabetes Mellitus Patients” was presented at the American College of Gastroenterology 2021 conference. The investigative team included Laurel Wood, MD, and colleagues variously affiliated with the University of Chicago. For the study, they reviewed chart data on patients under 21 years old who were diagnosed with both T1DM and celiac disease, and treated at University of Chicago. Data included patient diagnosis, pathologic results, serologic test results, and patient demographics. In all, 52% of patients were female, 86% were white, 5% were Hispanic or Latino, 5% were Asian, 3% were black, and 6% were of unknown ethnicity. The team found a total of sixty-three patients diagnosed with T1DM and celiac disease. Seven children diagnosed with T1DM who initially had negative celiac screening results became positive up to 7.25 years later, which shows the importance of regular screening. Typical T1DM patients received first celiac disease screening within an average of 2.29 years after their T1DM diagnosis. The diagnoses of celiac disease included sixteen cases by serology alone (16), thirty-nine by duodenal biopsy (39), and eight others. TTG and EMA showed a strong correlation, while high DGP IgA and DGP IgG were not necessarily the best indicators or duodenal mucosal damage in patients with T1DM. To more fully understand the usefulness of celiac disease antibodies in predicting severity of disease in T1DM children, the team suggests studies with larger samples of T1DM patients with Marsh scores less than 3 and increased celiac disease serology. They found that people with type 1 diabetes mellitus (T1DM) face a risk of developing celiac disease that is five to seven times higher than the general population. Read more at hcplive.com
  10. Celiac.com 09/29/2021 - My endocrinologist suspects I have celiac sprue. I am 47 years old and have had type 1 diabetes since I was 11 years old. HOWEVER...long before the diabetes I experienced severe cramping episodes for which my physicians could never identify a cause. I learned to live with it for 36 years. I am insulin resistant, and “carbohydrate sensitive”, and with the help of a highly competent endocrinologist, have gone from 231 lbs to 177 with an ultimate goal of 135 lbs. I went on an insulin infusion pump in May. The well-meaning diabetes educator was “instrumental” in setting the controls to my pump with the “idea” that I need not go below 120 grams of carbohydrate daily for weight and blood glucose management. As a result I have gained 9 lbs since May, and am working my pump settings back down with the idea of eating 55 - 60 grams of complex carbohydrates daily. My insulin usage is about 67% of what it used to be, yet I can’t take this additional weight off. I was “taught” to eat crackers and juice for low blood glucose level management. All of this paints a picture of my situation today. A few weeks ago I talked to my endocrinologist about my cramping issues. After a boat-load of vials of blood she has arrived at the suspicion that I may have celiac sprue. Since she said that, I have read many articles about this disorder and it seems to fit. It is likely the cause of my lifetime of cramping issues. My mother had “colitis”, and so does my sister. During times of distress they experience intense cramping paired with dizziness, nausea, weakness, and sometimes headaches. Sure sounds like a food issue to me. I try to maintain a 1000 calorie diet. Sometimes (especially if I have had gluten-laden foods, now that I look back and think about it...) I just about starve to death!!!! I am trying to adhere to the South Beach diet. I will be going for a G.I. consult in the near future. As a result of Ron Hoggan’s article in the Spring 2006 edition of this newsletter regarding the low incidence of celiac disease diagnoses with obese people,  I now have hope that I can lose the pain AND the weight with proper diet and exercise. Today I shall try to bear in mind the many great kindnesses that God has done for me, and ignore the relatively insignificant dis-pleasures in my life. A month later: I have stopped eating gluten with a few rare exceptions when my blood glucose levels are low and only glutinous foods are available. The interesting thing is, since I stopped eating the darned stuff, the cramps have ceased!  That seals it for me!! 
  11. Celiac.com 05/10/2021 - A top physician in Turkey recently warned that rates of autoimmune diseases like Type 1 diabetes and celiac will likely rise in the wake of the coronavirus pandemic. Autoimmune diseases, including thyroid issues, happen when the body attacks its own tissues. The coronavirus causes the body's immune system to produce “attacking” antibodies. Because "[v]iruses serve as a mechanism that pull the trigger for autoimmune diseases,” an increase in rates of autoimmune diseases was unavoidable after a year of pandemic," says Professor Tufan Tükek, head of the Faculty of Medicine at Istanbul University. Lingering COVID-19 symptoms Impede Autoimmune Disease Management Ongoing coronavirus symptoms have been a problem for numerous recovered patients, and can impair the management of autoimmune diseases. For example, in diabetes patients, studies show that the symptoms influence blood sugar levels, and impede its management by causing fatigue and memory issues. In March 2020, Istanbul University became one of the first institutions in the country to establish an observation center for recovered coronavirus patients. Since then, they have monitored nearly 4,000 patients. Professor Tükek says that, in the early days of the pandemic, diarrhea was the main "long COVID-19" symptom, and then, after a second COVID-19 wave last summer, their team began seeing more memory issues and hair loss. Lately, Tükek said, they are seeing more cases of blood clots. COVID-19 Symptoms Can Linger for Months Dr. Huzeyfe Arıcı, a physician working at the observation center, said that COVID-19 symptoms can linger for up to eight weeks, in many cases. “We have patients suffering from back pain that long, something that cannot be cured by painkillers. We also see an increasing number of cases with memory lapses,” he stressed. With COVID-19 survivors numbering in the millions, it is an open question as to how many will be affected by lingering symptoms, for how long, and what can be done to help them. The idea that coronavirus could increase rates of diabetes, celiac or other autoimmune conditions is bit alarming. Quantifying and describing the problems and then creating a way to address them is crucial. Look for more information as other observation centers share their observations on COVID-19 survivors. Read more in Dailysabah.com
  12. Celiac.com 02/13/2021 - I was interviewed for a national diabetes magazine the other day. They wanted to know how a diet such as the Specific Carbohydrate Diet would be for diabetes sufferers, especially since, in Australia, 10% of diabetics are also diagnosed with celiac disease. For diabetics the all important question is how carbohydrates affect their blood sugar level, and that the recommended foods have a low Glycemic Index. The Glycemic Index (GI) is a measurement of the type of carbohydrates in a particular food, and how fast 50 grams of this carbohydrate raises blood glucose levels (and consequent insulin secretion and effects produced by the pancreas) as it is digested. It is also important to consider the Glycemic Load of foods. For those of you who aren't familiar with it, the Glycemic Load was devised to make the Glycemic Index useful in the real world. The problem with the Glycemic Index is that the tests use 50 grams of carbohydrate from the food being tested. On a practical level, that means they test a plateful of spaghetti, but a truckload of cucumbers! It doesn't take into account how food is eaten in the real world, making benign foods seem damaging. The Glycemic Index is the measurement of how rapidly a given carbohydrate food is absorbed, and therefore how fast it spikes blood sugar. In general, a rapid rise in blood sugar triggers a large insulin release. The Glycemic Load is the Glycemic Index multiplied by the actual number of grams of carbohydrate eaten. Ten or less is a low Glycemic Load—11 to 20 is a medium load, and anything over 20 is high. Take carrots. Carrots have a high Glycemic Index for a vegetable—around 50. But do you know how many carrots you'd have to eat to get fifty grams of carbohydrate? More than fifty! The carbohydrate content of eating two whole carrots with a meal is too small to cause a significant rise in blood sugar levels. Oatmeal, on the other hand, has about the same GI as carrots, but a one cup serving of cooked oatmeal has 25 grams of carbohydrate, for a Glycemic Load of 12.5 in contrast to say 5 baby carrots which has 4 grams of carbohydrate and a Glycemic Load of 2—very low. So how do the foods allowed on the Specific Carbohydrate Diet rate, in regard to the GI and GL? Is this a good thing for diabetics and everyone else wanting to be healthier? The Specific Carbohydrate Diet is based on ‘Simple Carbohydrate Foods' or rather monosaccharides which are the single molecule carbohydrates which need no enzyme to be digested. Carbohydrate foods naturally divide themselves into two groups: 1. starches and refined sugars, and 2. everything else. It's the concentration of carbohydrates in the starches and refined sugars that makes them a problem to those with bowel disease and/or diabetes. The specific carbohydrates allowed on the diet and used in the Healing Foods cookbook are the ones that are in most low GI foods. These foods are simple fresh foods, such as fresh fruits and vegetables, some low starch pulses, nuts, meats, cheeses and yogurt. Even the baked goods which are sweetened with honey are acceptable as the almond meal used instead of the wheat flour contain monounsaturated fats which slows the absorption rate of glucose from the honey into the bloodstream. Considering all these factors, diabetics, digestive disease sufferers, and generally everyone who wants to live a more energetic and healthy life should be able to benefit from the recipes in Healing Foods: Cooking for Celiacs, Colitis, Crohn's and IBS.
  13. Celiac.com 09/28/2020 - Biomedical researchers Tuulia Hyötyläinen and Matej Orešič, at Örebro University, have published two studies connecting highly fluorinated chemicals to autoimmune diseases in children. In the studies, Hyötyläinen and Orešič, link per- and polyfluoroalkyl substances (PFAS's) – with type 1 diabetes and celiac disease, or gluten intolerance in children. PFAS are man-made chemicals. There are about 5,000 PFAS, and they are used in numerous consumer products, such as furniture, adhesives, food packaging, coatings in clothing, and even fire-fighting foam. In the celiac disease study, Hyötyläinen and Orešič show a connection between PFAS and celiac disease, by demonstrating that "high exposure to PFAS in the womb and in first years of life can accelerate the development of celiac disease in children," says Hyötyläinen. The studies appear in Environment Research. Their conclusions are based on analysis of the metabolization of small molecules in the body, and analysis of PFAS, coupled with a system's biology approach to integration of complex study data, gathered from expectant mothers and children. "Exposure to harmful chemicals in early life, including prenatally, may offer an explanation for the changing incidence of these autoimmune diseases in developed countries and can be connected to other health risks," says Matej Orešič. In Sweden and other Nordic countries, as in many other modern countries, type 1 diabetes is one of the most common chronic diseases among children, with cases rising sharply in the last few decades. Interestingly, the incidence curve has flattened in the last several years, which may result from stricter PFAS regulation in recent years. Researchers know that some children have genes that increase their likelihood of developing type 1 diabetes, but only about one in ten of them actually go on to develop diabetes. This strongly indicates an environmental factor as a potential trigger for autoimmune disease development. Both viral infections and diet have been singled out as potential triggers. In their recent work, Hyötyläinen and Orešič describe how PFAS impacts lipid metabolism and risk of type 1 diabetes in new-born children. Their study data shows that these chemicals are easily passed from exposed expectant mothers to the fetus. Moreover, it is known that "children exposed to the high levels of PFAS during the prenatal stage have a certain lipid profile...associated with an increased risk for type 1 diabetes and the development of the disease in children," explains Orešič. The team's findings are confirmed by another clinical study on children at-risk for type1 diabetes, along with two studies on experimental models of type 1 diabetes in mice. This is not the first study to show links between exposure to chemicals and the development of celiac disease, and other certain auto-immune conditions. The implications of the research for the understanding of type 1 diabetes and celiac disease in children remains unclear, as does information on safe or unsafe levels of PFAS exposure in humans. Certainly further study and consideration of the issue by researchers and clinicians is welcome. Stay tuned for more on the role of pesticide and chemical exposure in the development of celiac disease and other auto-immune conditions, including type 1 diabetes. Read more at Medicalxpress.com
  14. Celiac.com 06/22/2020 - Since 2004 data collected prospectively by The Environmental Determinants of Diabetes in the Young (TEDDY) study group has helped researchers to better understand T1D, and associated autoimmune conditions, like celiac disease. TEDDY is funded by the National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK). Celiac disease and T1D share a number of genetic factors. Seeking to explain why some children with high-risk genes develop T1D or celiac disease, while most do not, the TEDDY team monitors study subjects for both T1D and celiac disease. TEDDY research has already shown that genetically predisposed children who eat gluten at, or above, certain levels in early childhood, had higher rates of celiac disease. "An interesting finding from TEDDY has been how early the autoimmune destruction of insulin-producing cells begins–often in the initial two years of life," said study TEDDY co-chair Marian Rewers, MD, PhD, a professor of pediatrics and medicine and executive director of the Barbara Davis Center for Diabetes at the University of Colorado School of Medicine. The TEDDY study follows infants with high T1D risk factors for 15 years to look for certain beta-cell autoantibodies and diabetes. TEDDY has also looked at biomarkers that indicate faster or slower progression to diabetes after autoimmune destruction begins. "While T1D and celiac disease share a lot of genetic characteristics, there are intriguing differences in the ways these diseases develop and progress," says Dr. Rewers, adding that "TEDDY research and discovery will help drive the "design of future trials to prevent both T1D and celiac disease." TEDDY is looking to uncover viruses and nutritional factors that work with genes to initiate destruction of the beta cells by the immune system, which is signaled by the appearance of islet autoantibodies. Ultimately, TEDDY investigators are looking to uncover a way to prevent both diabetes and celiac disease in children. The latest information from TEDDY highlights potential "triggers" for the autoimmune process that generates type 1 diabetes (T1D), and how those triggers engage in children with with genetic risk factors for T1D. That information is highlighted in the "Update from the TEDDY Study" symposium today at the American Diabetes Association's (ADA's) 80th Virtual Scientific Sessions. Among TEDDY's latest findings are two new papers, Longitudinal Metabolome-Wide Signals Prior to the Appearance of a First Islet Autoantibody in Children Participating in the TEDDY Study; and Distinct Growth Phases in Early Life Associated With the Risk of Type 1 Diabetes. Read the original press release at PRNewswire.com. Stay tuned for more on TEDDY and related stories.
  15. To All, I recently came across this new research that show Environmental Toxins can increase the risks of Celiac disease in Children? Could this also be happening in Adults? https://medicalxpress.com/news/2020-09-highly-fluorinated-chemicals-diabetes-coeliac.html Have they found an Environmental trigger for Celiac disease and if so what can people do about it if anything? I know Celiac.com will write a more exhaustive article about it soon.....I just wanted to get it out there for people to comment on and provide feedback too! I hope this is helpful but it is not medical advise. Posterboy,
  16. Celiac.com 06/09/2020 - What can science tell us about celiac disease screening rates and glycemic outcomes of a gluten-free diet in patients with type 1 diabetes who are asymptomatic for celiac disease? A team of researchers recently set out to assess the issue and get some answers. The research team included Farid H. Mahmud, Antoine B.M. Clarke, Kariym C. Joachim, Esther Assor, Charlotte McDonald, Fred Saibil, Heather A. Lochnan, Zubin Punthakee, Amish Parikh, Andrew Advani, Baiju R. Shah, Bruce A. Perkins, Caroline S. Zuijdwijk, David R. Mack, Dror Koltin, Emilia N. De Melo, Eugene Hsieh, Geetha Mukerji, Jeremy Gilbert, Kevin Bax, Margaret L. Lawson, Maria Cino, Melanie D. Beaton, Navaaz A. Saloojee, Olivia Lou, Patricia H. Gallego, Premysl Bercik, Robyn L. Houlden, Ronnie Aronson, Susan E. Kirsch, William G. Paterson, and Margaret A. Marcon. They are all affiliated with the American Diabetes Association. The team conducted celiac disease screening on asymptomatic patients from 8 to 45 years of age. To assess changes in HbA1c, they randomly assigned biopsy-confirmed celiac disease patients to a gluten-free diet or gluten-containing diet (GCD), along with one year of glucose monitoring. Adults tested positive for celiac disease antibodies more often than children with lower rates of prior celiac disease screening. Twenty-seven subjects went on the gluten-free diet, while twenty-four followed the gluten-containing diet. The team saw no HbA1c differences between the groups, though gluten-free patients showed more substantial glucose increases after meals. Celiac disease is common in asymptomatic patients with type 1 diabetes, and the team advises clinicians to be vigilant about starting those patients on a gluten-free diet. Read more in Diabetes Care 2020 May; dc191944.
  17. Hi All, My son is a 28 year old type 1 diabetic with celiac disease. He is currently in the hospital after becoming very ill 2 days ago, then moved to ICU. They have not determined exactly what the cause of his illness, but it sounds like they are leaning toward gastroparesis. I am doing what I can to research and be informed so I can be a support system for him. He has really been struggling, especially the past couple of months, with nerve pain down his legs, anxiety and panic attacks, fatigue, weight loss, light-headedness, very pale, etc. Are these symptoms that any of you had prior to your diagnosis? Deep down I have felt there was some underlying cause. Any input would be helpful. It is really hard right now because of Covid-19, I can't even visit him. Thanks!
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