Ok I've thought about this more deeply and now I have all kinds of new questions.
1. Does the fact that the body CAN make an antibody against gluten inherently mean the body is coded to recognize gluten as an antigen (as in, you are born thinking that gliadin is an antigen)? Or does it mean that the body taught itself later on how to make an antibody against gliadin?
2. When the immune system produces antibodies against gliadin, is this a reaction of a "confused" immune system which mistakenly thinks that gliadin is an antigen? Is the immune system wrong about this? What tells the immune system that something is an antigen?
3. The immune system, as we all know, does make mistakes. For example, when it makes killer cells that attack the "self" cells in an autoimmune response, I would consider that to be a confused mistake that the immune system makes. But how did the immune system get confused and think that the "self" cells were antigens or pathogens?
4. How does the immune system make antibodies against a cell it thinks is an antigen or a pathogen? Does this information need to be pre-coded into our DNA, or can the immune system make antibodies against anything and everything that it thinks is an invader? What are the restrictions and limitations on this?
Ok so I've been thinking about this. If your body is making AGA-IgG or AGA-IgA, doesn't that mean that your body is having an immune reaction to gluten?
AGA-IgG and AGA-IgA are antibodies. Antibodies are products that the body makes in response to what the body thinks is a foreign invader that needs to be killed off. It's an immune response.
If your body is creating ANY sort of immune response to gluten, doesn't that mean you need to stop eating gluten? Long-term low-level immune activity over the long-term could lead to autoimmune diseases or put the body in a state of oxidative stress, right?
What percentage of the population makes AGA-IgG/AGA-IgA antibodies?