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    Is the Global Gluten-free Pet Food Market Set to Explode?


    Jefferson Adams


    • Is gluten-free pet food the next great investment?


    Image Caption: Could gluten-free pet food be the next big thing? Photo: CC--Franchise Opportunities.

    Celiac.com 08/04/2017 - Industry analysts are projecting the global market for gluten-free pet foods to enjoy growth of up to 25% a year over the next decade. Across numerous industries, a shift from products containing gluten to gluten-free products is creating major potential for manufacturers.


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    The latest market report from Persistence Market Researchers, titled Global Gluten-free Pet Food Market: Drivers and Restraints, projects double-digit growth in gluten-free pet food markets through 2025.

    The report offers market information and analysis on all segments of the global gluten-free pet food market broken down by type, flavor, specification, form, and distribution channel. Types include natural and added additives, while flavor types are further divided into chicken, beef, fish, and other red meat and white meats.

    Specification covers the type of pet, such as food for cats, for dogs, for birds, for pocket animals, and others. The report breaks down each of these categories.

    In terms of distribution channel, the global gluten-free pet food market report includes information on e-commerce, supermarkets, retail shops, exclusive pet shops, and others. Form type includes information on dry and wet pet food market segments.

    Gluten-free pet food is a new segment in the pet food industry, and has strong potential to displace regular pet foods.

    North America currently leads the world in gluten-free pet food production. Currently, there are no gluten-free pet food manufacturers in Europe.

    Meanwhile, North America and Europe are currently the largest consumers of gluten-free pet food products followed by Asia Pacific.

    A Sample of this Report is Available Upon Request at: PersistenceMarketResearch.com


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    Guest Tania J Malven

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    Our dog was diagnosed with inflammatory bowel syndrome at age 2. Rx: diet free of wheat, corn, soy, dairy, beef, chicken and eggs. He did very well for the next 11.5 years!

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    My dog doesn't have celiac, I do. But to avoid cross contamination issues and keep life simple, my dog gets a grain free food. It's simply one less thing to worry about.

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    Jefferson Adams
    02/01/2011 - Imagine having a dog that was specially-trained to sniff out even the tiniest amounts of gluten in food and warn you ahead of time. There are scores of people with celiac disease severe enough that the slightest trace of gluten can make them painfully ill. Hollie Scott is one of them. Scott is a University of Missouri College of Veterinary Medicine student is also lucky to have her dog Elias is a champion Beauceron and a gluten-detecter extraordinaire. The handsome Beauceron comes from a 400-year-old breed that became almost extinct serving as messenger dogs in Europe during two world wars. Even though he is just only 2 years old, Elias is the first male Beauceron to receive the title AKC Grand Champion. His full title is: GCH CH Elias Mes Yeux Vigilants RN. But Elias' regular job is working as a gluten-detection service dog for his twenty-two year old owner, Scott, a first-year student in the program.
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    Jefferson Adams
    Celiac.com 05/04/2016 - First, the good news. Canary seed, commonly used as feed for its namesake yellow birds has been approved as gluten-free and fit for human consumption in Canada.
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    Source:
    techmalak.com

    Jefferson Adams
    Celiac.com 08/04/2016 - With all the hype about gluten-free diets going around, it's only natural that dog owners might wonder about potential benefits of a gluten-fee diet for their pet. Before rushing out and spending money on a bunch of new gluten-free grub for Fido, it's wise to first ask a few questions.
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    Jefferson Adams
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    Maybe one day dogs like Zeus will be much more common.
    See more at NBCNews.com

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    Jefferson Adams
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    Dr. Ron Hoggan, Ed.D.
    Celiac.com 06/15/2018 - There seems to be widespread agreement in the published medical research reports that stuttering is driven by abnormalities in the brain. Sometimes these are the result of brain injuries resulting from a stroke. Other types of brain injuries can also result in stuttering. Patients with Parkinson’s disease who were treated with stimulation of the subthalamic nucleus, an area of the brain that regulates some motor functions, experienced a return or worsening of stuttering that improved when the stimulation was turned off (1). Similarly, stroke has also been reported in association with acquired stuttering (2). While there are some reports of psychological mechanisms underlying stuttering, a majority of reports seem to favor altered brain morphology and/or function as the root of stuttering (3). Reports of structural differences between the brain hemispheres that are absent in those who do not stutter are also common (4). About 5% of children stutter, beginning sometime around age 3, during the phase of speech acquisition. However, about 75% of these cases resolve without intervention, before reaching their teens (5). Some cases of aphasia, a loss of speech production or understanding, have been reported in association with damage or changes to one or more of the language centers of the brain (6). Stuttering may sometimes arise from changes or damage to these same language centers (7). Thus, many stutterers have abnormalities in the same regions of the brain similar to those seen in aphasia.
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    Sources:
    1. Toft M, Dietrichs E. Aggravated stuttering following subthalamic deep brain stimulation in Parkinson’s disease--two cases. BMC Neurol. 2011 Apr 8;11:44.
    2. Tani T, Sakai Y. Stuttering after right cerebellar infarction: a case study. J Fluency Disord. 2010 Jun;35(2):141-5. Epub 2010 Mar 15.
    3. Lundgren K, Helm-Estabrooks N, Klein R. Stuttering Following Acquired Brain Damage: A Review of the Literature. J Neurolinguistics. 2010 Sep 1;23(5):447-454.
    4. Jäncke L, Hänggi J, Steinmetz H. Morphological brain differences between adult stutterers and non-stutterers. BMC Neurol. 2004 Dec 10;4(1):23.
    5. Kell CA, Neumann K, von Kriegstein K, Posenenske C, von Gudenberg AW, Euler H, Giraud AL. How the brain repairs stuttering. Brain. 2009 Oct;132(Pt 10):2747-60. Epub 2009 Aug 26.
    6. Galantucci S, Tartaglia MC, Wilson SM, Henry ML, Filippi M, Agosta F, Dronkers NF, Henry RG, Ogar JM, Miller BL, Gorno-Tempini ML. White matter damage in primary progressive aphasias: a diffusion tensor tractography study. Brain. 2011 Jun 11.
    7. Lundgren K, Helm-Estabrooks N, Klein R. Stuttering Following Acquired Brain Damage: A Review of the Literature. J Neurolinguistics. 2010 Sep 1;23(5):447-454.
    8. [No authors listed] Case records of the Massachusetts General Hospital. Weekly clinicopathological exercises. Case 43-1988. A 52-year-old man with persistent watery diarrhea and aphasia. N Engl J Med. 1988 Oct 27;319(17):1139-48
    9. Molteni N, Bardella MT, Baldassarri AR, Bianchi PA. Celiac disease associated with epilepsy and intracranial calcifications: report of two patients. Am J Gastroenterol. 1988 Sep;83(9):992-4.
    10. http://ezinearticles.com/?Food-Allergy-and-Stuttering-Link&id=1235725 
    11. http://www.craig.copperleife.com/health/stuttering_allergies.htm 
    12. https://www.celiac.com/forums/topic/73362-any-help-is-appreciated/
    13. Ford RP. The gluten syndrome: a neurological disease. Med Hypotheses. 2009 Sep;73(3):438-40. Epub 2009 Apr 29.
    14. Hadjivassiliou M, Gibson A, Davies-Jones GA, Lobo AJ, Stephenson TJ, Milford-Ward A. Does cryptic gluten sensitivity play a part in neurological illness? Lancet. 1996 Feb 10;347(8998):369-71.

    Jefferson Adams
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    They also noted a higher frequency of surface CD3(−) IELs in cases with clonal TCR-GR, but the PCP pattern showed no associations with any clinical or pathological feature. 
    Repeat biopsy showed that the clonal or PCP pattern persisted for up to 2 years with no evidence of RCDII. The study indicates that better understanding of clonal T cell receptor gene rearrangements may help researchers improve refractory celiac diagnosis. 
    Source:
    Journal of Clinical Pathologyhttp://dx.doi.org/10.1136/jclinpath-2018-205023

    Jefferson Adams
    Celiac.com 06/13/2018 - There have been numerous reports that olmesartan, aka Benicar, seems to trigger sprue‐like enteropathy in many patients, but so far, studies have produced mixed results, and there really hasn’t been a rigorous study of the issue. A team of researchers recently set out to assess whether olmesartan is associated with a higher rate of enteropathy compared with other angiotensin II receptor blockers (ARBs).
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    Source:
    Alimentary Pharmacology & Therapeutics

    Jefferson Adams
    Celiac.com 06/12/2018 - A life-long gluten-free diet is the only proven treatment for celiac disease. However, current methods for assessing gluten-free diet compliance are lack the sensitivity to detect occasional dietary transgressions that may cause gut mucosal damage. So, basically, there’s currently no good way to tell if celiac patients are suffering gut damage from low-level gluten contamination.
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    Source:
    Gut. 2017 Feb;66(2):250-257.  doi: 10.1136/gutjnl-2015-310148.