wellerval replied to Almedingen's topic in Celiac Disease - Pre-Diagnosis, Testing & SymptomsHi Barbara, I am wondering if you have had Vitamin D testing done, as that can lead to Fibromyalgia-like pains, kind of like calcium is being pulled out of your bones.... The test you want is called 25(OH)D. Most lab's stated reference range is 32 - 100 ng/mL, but most of the experts suggest 55-70 ng/mL for optimal health. If you are below 40 ng/mL, you may want to consider supplementing about 10,000 IU/day with 1,500 mg of calcium. This would bring your D level up within a few weeks, I would think. We use a liquid D supplement that is 2,000 IU per drop, making it very easy to take. It is made by Rx Vitamins, but I am sure there are other brands out there. Best of luck to you, Leslie
PLEASE read the following article on the relationship between h.Pylori (which can be cured) and MALT Lymphoma. A non-prescription supplement that has been shown to eradicate h. Pylori is called Pepzin GI. I have no affiliation with the supplement manufacturer. Google Pubmed.org and zinc carnosine to see articles related to the Japanese study that showed success of Pepzin GI. I was researching gastroparesis for my sister-in-law and came across your website. I have seen lots of links to iron, heme, carbon monoxide, oxytocin and h. pylori with regard to gastroparesis and wonder if the low iron experienced by Celiac could be a result of h. pylori infection. H.Pylori takes iron for its own needs, thereby depriving the body of sufficient iron. I believe all celiac sufferers should have h. pylori testing. Rinsho Byori. 2009 Sep;57(9):861-9. [Crucial roles of Helicobacter pylori infection in the pathogenesis of gastric cancer and gastric mucosa-associated lymphoid tissue (MALT) lymphoma] [Article in Japanese] Ota H, Asano N, Yamauchi K, Akamatsu T. Department of Biomedical Laboratory Sciences, School of Health Sciences, Shinshu University School of Medicine, Matsumoto 390-8621, Japan. firstname.lastname@example.org Helicobacter pylori (H. pylori) is a gram-negative helical rod that colonizes human gastric mucosa. Its discovery has opened up new opportunities regarding the understating and management of gastrointestinal disorders. In humans, infection with H. pylori has been established as a major cause of chronic gastritis and peptic ulcer, and is important in the pathogenesis of gastric cancer and gastric mucosa-associated lymphoid tissue (MALT) lymphoma. Bacterial and host factors determine the outcome of H. pylori infection. The eradication of H. pylori can, therefore, contribute to the treatment and prevention of these diseases. H. pylori infection plays a critical role in gastric carcinogenesis through two major pathways: the indirect action of H. pylori on gastric epithelial cells through inflammation, and the direct action of the bacteria on epithelial cells through the induction of protein modulation and gene mutation. MALT lymphoma is a common low grade B-cell lymphoma arising from a background of chronic inflammatory disease at a number of mucosal sites. Those originating in the stomach are causatively linked to H. pylori infection, and eradication of the bacterium with antibiotics leads to the long-term complete regression of lymphoma. t (11;18)/API2-MALT1 and t(1;14)/IGH-BCL10 are specifically associated with the gastric MALT lymphoma entity, and the oncogenic products of these translocations have been shown to target a common molecular pathway, i.e., the nuclear factor-kappaB pathway. This paper reviews recent advances in our understanding of the association of H. pylori infection with gastric cancer and gastric MALT lymphoma and the molecular genetics underlying tumor development.