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Showing results for tags 'gastritis'.
Found 13 results
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Hello! I was recently diagnosed with celiacs and mild chronic gastritis was noted on my EGD as well. My GI did not even bring up the gastritis and when I asked she brushed it off. I really want to heal my gut completely not just from celiacs but also the gastritis. Did anyone else have this at diagnosis? Were you able to treat it? Was it associated with celiacs or something entirely different? Thank you!!
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Hello everyone! I've been suffering celiac symptoms for about 7 years, was diagnosed through blood a year ago, and confirmed through endoscopy in February 2024. I am HIGHLY ALLERGIC (epi-pen worthy) to dairy and have been since 2011, and I've been gluten free since 2023 except for the minor forced glutening before my scope. During that procedure it was also found that I have chronic gastritis and chronic esophagitis--they believe this is related to the celiac inflammatory response. I am currently in the midst of a gastritis attack, and I was hospitalized due to a minor bleed in my system. That being said, I already have a strict diet. I am gluten and dairy free. I never eat out and I am so careful. However, my new rules for the next 4-6 months are that I MUST cut out: -caffeine, carbonation, citrus, tomato or anything tomato-based, onion, high-fat/oily foods, anything spicy. I want...to cry. I am a foodie at heart. I love food. I love being in the kitchen with my family. I love to eat. Lately it's been touch and go because I've been in so much pain and having so many symptoms but to see it laid out so plainly like this; it makes me quite sad. I could easily cope if it were 2-4 weeks but at half a year, it seems like adapting to a new lifestyle. Does anyone have any tips or tricks for coping with such a change? -
Celiac.com 05/23/2023 - In recent years, there has been a rapid decline in the cases of Helicobacter pylori gastritis, particularly in developed countries. However, amidst this decline, an intriguing phenomenon has emerged: Helicobacter pylori-negative chronic gastritis. This condition is marked by chronic inflammation of the stomach lining in the absence of the notorious Helicobacter pylori bacterium, and has gained increasing recognition among experts as an important histological finding. Despite the growing awareness of this condition, the rates and clinical significance of Helicobacter pylori-negative chronic gastritis in children remain poorly studied. To shed more light on this condition, a team of researchers set out to learn more about this peculiar type of gastritis in the younger population. The research team included Anni Virkkula, Laura Kivela, Pauliina Hiltunen, Antti Sotka, Heini Huhtala, Kalle Kurppa, and Marleena Repo. They are variously affiliated with theTampere Centre for Child, Adolescent and Maternal Health Research, Faculty of Medicine and Health Technology, Tampere University, Tampere, Finland; the Celiac Disease Research Centre, Faculty of Medicine and Health Technology, Tampere university, Tampere; the University of Helsinki and Helsinki University Hospital, Children's Hospital, and Paediatric Research Center, Helsinki; the Department of Pediatrics, Tampere University Hospital, Tampere; the Department of Pediatrics, South Karelia Central Hospital, Lappeen-ranta; the Faculty of Social Sciences, Tampere University, Tampere; the University Consortium of Seinajoki and Seinajoki Central Hospital, Seinajoki, Finland; and the Department of Pediatrics, Central Finland Central Hospital, Jyvaskyla, Finland. The team began their investigation by gathering data from 1,178 consecutive children who underwent diagnostic esophagogastroduodenoscopy (EGD). By comparing the baseline characteristics and long-term outcomes of children with active and inactive Helicobacter pylori-negative chronic gastritis, as well as those with normal gastric histology, they sought to discern patterns and draw meaningful conclusions. They then tracked follow-up data for up to 13 years. What the team discovered was nothing short of remarkable. Helicobacter pylori-negative chronic gastritis, it turns out, is a rather common finding in children undergoing EGD. Intriguingly, the active type of this form of gastritis was found to be particularly associated with Crohn's disease, an inflammatory bowel condition, while the inactive form exhibited links to celiac disease. In addition to shedding light on these associations, the study uncovered another fascinating revelation—Helicobacter pylori-negative chronic gastritis can serve as a predictor of gastrointestinal diagnoses, most notably inflammatory bowel disease and celiac disease. This finding carries significant implications for early detection and subsequent management of these conditions. Furthermore, the researchers examined the long-term prognosis of patients diagnosed with Helicobacter pylori-negative chronic gastritis who did not receive an initial diagnosis. Encouragingly, they found that the prognosis for these individuals is generally favorable. With valuable insights into prevalence, clinical significance, and potential implications for gastrointestinal health, this study marks an important evolution in our understanding of Helicobacter pylori-negative chronic gastritis. Read more in the Journal of Pediatric Gastroenterology and Nutrition 74(5):p 949-955, May 2022 -
Hello all. I had a routine liver enzyme test last December which came back elevated. My GP did loads of further tests one of which was for celiac which came back positive. I was later found to have hemochromatosis which appears to be the cause of the elevated liver enzymes. Due to the positive celiac blood test I was put on the waiting list for an endoscopy which I had done 2 days ago. The doctor who did the procedure took 6 biopsies & various photos. He spoke to me after the procedure & explained that I had mild gastritis / duodenitis & the report also says patchy erythematous. I asked him if celiac disease could cause this & his reply was 'possibly'. That is all I was told & i suffer from health anxiety so I am panicking a bit to be honest in case it's something serious. I don't drink, smoke & only take NSAID when needed. Can these things be caused be celiac disease? -
Celiac.com 12/05/2022 - Helicobacter pylori (H. pylori) is one possible cause for dyspepsia. However, dyspeptic symptoms are also common in H. pylori negative gastritis. Understanding the underlying causes could help improve treatment strategies for dyspepsia and chronic gastritis. A team of researchers recently set out to assess rates of dyspeptic symptoms in patients with H. pylori negative chronic gastritis, and explore any potential role played by autoimmunity. The research team included Noémi Zádori, Dávid Németh, Levente Frim, Nóra Vörhendi, Lajos Szakó, Szilárd Váncsa, Péter Hegyi, and József Czimmer. They are variously affiliated with the Institute for Translational Medicine, Medical School, University of Pécs, Pécs, Hungary; the János Szentágothai Research Centre, University of Pécs, Pécs, Hungary; the Centre for Translational Medicine, Semmelweis University, Budapest, Hungary; the Division of Gastroenterology, First Department of Medicine, Medical School, University of Pécs, Pécs, Hungary For their retrospective study, the team included data from patients with H. pylori negative chronic gastritis. They excluded: patients with acute gastritis; reactive gastropathy; subjects without serology test results; H. pylori positivity; presence of atrophy, intestinal metaplasia (IM), gastroesophageal reflux disease (GERD), ulcer, or cancer. The researchers assessed the following endpoints: the rate of dyspepsia-like symptoms; links between dyspepsia and autoimmune disease-related seromarker positivity (AISP); frequency of other symptoms in chronic gastritis and its association with AISP; and location of the inflammation and its association with AISP. For their study, the team included 175 from a total of 285 patients. A total of ninety-five of these 175 patients, nearly 55%, experienced dyspeptic symptoms. Overall, AISP was more common in these dyspeptic patients, especially celiac seropositivity, anti-neutrophil cytoplasmic antibody (ANCA) and anti-Saccharomyces cerevisiae antibodies (ASCA) positivity. The team found no significant connections with other tested autoimmune (AI)-related antibody positivity. Having positive seromarkers for autoimmune diseases in chronic gastritis may make people susceptible to dyspeptic symptoms, and may be the cause of many cases of dyspepsia. According to their findings: "From a total of 285 patients, 175 were included in this study. Among these patients, 95 experienced dyspeptic symptoms (54.29%) and were associated more with AISP (p = 0.012), especially with celiac seropositivity (p = 0.045), anti-neutrophil cytoplasmic antibody (ANCA) and anti-Saccharomyces cerevisiae antibodies (ASCA) positivity (p = 0.043). A significant association was not found with other tested autoimmune (AI)-related antibody positivity." The data suggest that additional studies will help to clarify whether antibody screening in patients with dyspepsia will help shrink the time for autoimmune disease diagnoses, and whether screening is economically and medically beneficial. Read more at dovepress.com -
I got diagnosed with gastritis due to excessive alcohol use and have continued to have the gastritis for the past two months. About a month ago I started having digestive issues and some symptoms of celiacs. The GI and my PCP have said that alcohol shouldnt be affecting my system anymore because I haven't drank in two months and the inflammation in the stomach should have healed so something else is causing it. Considering celiacs or IBS. But what I'm wondering is did the excessive alcohol use trigger the celiacs to appear? I really hope not because this is really killing me and I'm thinking back to all the moments I could have chosen to not drink. Please help! -
I’m still trying to get to the bottom of what’s causing my gastritis... my medical team doesn’t even Know, it could be stress or gluten or both.. I’ve been gluten free three weeks now and just now getting some relief from my gastritis pain.. I’ve had all sorts of tests done, all negative of other things that cause gastritis..and another thing is only the antrum(lower stomach near small intestine is affected by it). They only saw it here as they took biopsies from my whole stomach. Anyone else had the same experience ? Does this ever get better? -
Hello,I was diagnosed about two weeks ago been gluten free about three now..at time of my endoscopy the biopsies finally showed celiacs but also showed mild chronic gastritis in my antrum(lower stomach) I don’t know if this can be connected or caused by my untreated celiacs or not?ive herd lots of mixed answers on this and know a lot of celiacs don’t experience this symptom but they’ve done tests for everything else that can cause gastritis which have all come out negative.it will cause me pain and burning sensation in my stomach and makes me not want to eat..if this is related to celiacs and anyone has also experienced this gastritis how long on gluten free diet did it take for your stomach lining to heal or feel better? What do you suggest to encourage healing ?ive really fallen in a depression because of the discomfort and new diagnoses, feeling like it’s my fault at times somehow. Also I had to do a gluten challenge in September to do second biopsy to try to diagnose celiacs and ever since then the gastritis has been bad. Also I feel my ppi nexium worsens my gastritis pain for some reason.
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Hi all, I am 19, Male and live inside the UK. I weigh around 10st and I am 6ft 4. To cut a long story short, I am not officially diagnosed with celiac disease but gluten causes me crippling pain to the point where I can't even walk, severe diarrhea and loss of appetite. The crippling pain literally disappeared within two days of going gluten free. I am now three weeks down the line and I feel almost back to normal except I feel nauseous all the time during the day. My symptoms goes as follows: Tenderness at the top of the abdomen Nausea 1 - 2 hours after eating (sometimes even after water), Usually lasts for a few hours Nausea is relieved/improves immediately after eating If I don't eat anything I have zero nausea Nausea is usually completely gone in the evenings (after 7-8 PM or so) Stomach pain/heartburn is more severe when I haven't eaten for a while or lying down I have had a blood tests for quite a few things such as liver function, thyroid, diabetes, Iron and B12 levels just to name a few. I have had a full body CT scan, all clear but had slight inflammation of the terminal ileum (bit connecting small intestine to colon). An ultrasound of the abdomen (including gallstones) which again was all clear. Urine tests were clear. I had these exact same issues, nausea and pain two years ago so had an MRI done. Showed exact same inflammation in terminal ileum as now so had a colonoscopy done which showed lymphoid hyperplasia (was put down to an infection). Symptoms went away during the summer holiday due what I believe a change of diet (as in I stopped consuming a baguette and muffin each day). I still didn't feel 100% since then but the nausea was so minimal I didn't even notice but I still felt drained, lethargic and had brain fog. I'll be having another colonoscopy on the 29th just to check for Irritable Bowel Disease because out of all the tests I have had done, the only thing that is abnormal is the terminal ileum in the CT scan. I have not had an endoscopy. Did anyone else experience nausea almost 24/7 except midnight or before breakfast? The Doctor suspects gastritis and so have been on 20mg of omeprazole for three days. I am literally begging for answers right now, as I've been bedridden since August (up until I went gluten free in the first week of December which now leaves the nausea) and should be enjoying my life at university but I am instead stuck at home feeling nauseated each day. I've been admitted to A&E three separate times due to severe pain and haven't seen anyone besides the people that live in my house or the doctors since August. It literally feels like I have been to hell and back so any ideas, literally anything will do. Finally, I am 2000% sure I am not being cross contaminated, using stainless steel pans and there is currently no gluten inside the house. Thank you for reading -
Celiac.com 07/20/2015 - Lymphocytic gastritis (LG) is an uncommon gastric disorder with varying symptoms and endoscopic manifestations. LG, along with two forms of H. pylori-negative gastritis [chronic active gastritis (CAG) and chronic inactive gastritis (CIG)], appears to be more common in patients with celiac disease, based on single-center studies. A team of researchers set out to compare the prevalence of LG, CAG and CIG among those with normal duodenal histology or non-specific duodenitis, and those with celiac disease, as defined by villous atrophy, Marsh 3. The research team included B. Lebwohl, P. H. R. Green, and R. M. Genta. The are variously affiliated with The Department of Medicine, Coeliac Disease Center, Columbia University College of Physicians and Surgeons, New York, NY, The Department of Epidemiology, Mailman School of Public Health, Columbia University, New York, NY, USA, the Miraca Life Sciences, Irving, TX, USA, and the Departments of Pathology and Medicine (Gastroenterology), UT Southwestern Medical Center, Dallas, TX, USA The team analyzed all concurrent gastric and duodenal biopsy specimens submitted to a national pathology laboratory during a 6-year period. They then performed multiple logistic regression to identify independent predictors of each gastritis subtype. They found that a total of 287,503 patients underwent concurrent gastric and duodenal biopsy, with an average age of 52, and most (67%) being female. Compared to patients with normal duodenal histology, LG was more common in partial villous atrophy (OR: 37.66; 95% CI: 30.16–47.03), and subtotal/total villous atrophy (OR: 78.57; 95% CI: 65.37–94.44). Celiac disease was also more common in CAG (OR for partial villous atrophy 1.93; 95% CI: 1.49–2.51, OR for subtotal/total villous atrophy 2.42; 95% CI: 1.90–3.09) and was similarly associated with CIG (OR for partial villous atrophy 2.04; 95% CI: 1.76–2.35, OR for subtotal/total villous atrophy 2.96; 95% CI: 2.60–3.38). From this study, the team concluded that lymphocytic gastritis is strongly associated with celiac disease, with increasing prevalence correlating with more advanced villous atrophy. Chronic active gastritis and chronic inactive gastritis are also significantly associated with celiac disease. Future research should measure the natural history of these conditions after treatment with a gluten-free diet. Source: Alimentary Pharmacology & Therapeutics Volume 42, Issue 2, pages 180–187, July 2015. DOI: 10.1111/apt.13249 -
Hi, I was diagnosed with celiacs after extreme stomach pain, irregular bowel movements and diarrhea. The extreme stomach pain was the main reason I really needed to find out what this was, and finally I had a doctor let me know i probably had Celiacs after a blood test. An endoscopy came back positive for celiacs as well. The extreme burning in my stomach went away within a week, however the pain persisted. Whenever I ate, it became really bad, even though i was extremely careful and made everything from scratch. I even are only fruit for a whole week to make sure I wasn't screwing up. Now its been over a year, and I still have the stomach pain come back after eating almost anything other than juicy fruit like watermelon/melons/grapes. I got tested for H pylori a year ago, came back negative. Got tested for blood in stool: negative. Went to doctors, they say they have no clue what it is. I have tested with different things. If I eat very fatty food it hurts a lot more than other food. If I have alcohol, the pain turns into a very dull ache the next day after waking up, and it goes away the day after completely without fail. If I fast for a day, the pain does not exist. It used to hurt, but in the last few months not anymore. If I dont eat after 6, the pain is completely gone until I eat the next day, and it can be anything, even a no fat food. If I eat something at it starts hurting more, drinking a lot of water makes it usually go away almost completely. But that uncomfortable feeling is still there, just the major pain goes away. Im going crazy here trying to figure this out. Im 22 years old, I want to enjoy my life. It also seems to go away with exercise. Any help would be great, thank you! -
Great news! I have have joined the “Peter club”! My villi are healed! The gluten free diet is working! I was diagnosed around 4/2013 with only a positive DGP IgA and a Marsh Stage IIIB (moderate to severe villi damage). My main symptom was anemia which I recovered from in just a few months with iron supplements. Minor symptoms persisted the first year as I discovered food intolerances (e.g. Xanthan Gum) and worked with known intolerances (lactose, garlic, mushrooms, eggs, etc). Many intolerances resolved (and some did not) as I healed. A year later, I was diagnosed with diabetes and further modified my diet (fewer carbs, more fats, less sugar, even from grains). I did well. I did not return to my GI and instead worked with my PCP. I never had my antibodies tested after my initial diagnosis to see if they were coming down (bad move). I did get a bone scan after I experienced some fractures and was checked for nutritional deficiencies. I stupidly assumed that my antibodies would always be elevated like my thyroid antibodies have been for 20 years. I strongly recommend annual check ups for celiacs. Over the years, My DGP IgA levels were off the chart tested after hidden gluten exposures. My symptoms were severe and were not consistent which prompted my visits to my new GI. I would recover, though it took months....like 6. In January 2017, I think I was glutened again (I had not eaten eaten out for a year except at 100% gluten-free restaurants ), and I ate mostly unprocessed foods. I had a tooth infection, tooth extraction, flu and a cold all in a span of one month. I developed chronic hives that last for six months. So, I went back to my GI (off the charts DGP IgA). My allergist blamed my Hashimoto’s or an undiagnosed autoimmune issue, my PCP just mentioned how I am always chronically inflamed. My GI offered an endoscopy, but I declined. Instead, I opted for a slightly modified Fasano diet. I did not give up coffee! Finally, I was still having abdominal issues, so I requested the endoscopy for the first week in January. Why is my DGP IgA always elevated? Where was I getting gluten? Heck, my hubby has been gluten-free for 17 years, so I know the diet. He was okay though there are some things he never eats and I do. Am I super sensitive or am I developing another AI issue? Maybe since I have other antibodies (thyroid) floating around, I should discard this test result (not much research for post diagnosis testing issues). I was driving myself crazy. So, I needed to know for sure what was going on. This time, my new GI had a newer scope. I was shown the results (photos) while in recovery. You could could see the villi as the magnification was that strong). I did have a stomach polyp which was removed and biopsied. The pathologist reported no damaged in my duodenum, and that the polyp was not cancer and no H. Pylori. He noted chronic gastritis (this explains my stomach pinching sensation and indigestion, etc.) I was advised to continue my gluten free diet. I assume my stomach is still healing as my antibodies are no doubt still elevated (off charts in 4/2017 and 80 in 11/2017) though hopefully lower than 80. I am now considering giving up coffee for a while and looking into foods that will help heal the gastritis since I react to so many different medications (anaphylactic) and each medication must be carefully monitored. Oddly, did not have gastritis when I was initially diagnosed. I am happy to be a part of the Peter club! I plan on staying on the modified Fasano diet until my stomach has healed (though I did eat some homemade (Xanthan Free) cookies over the holidays). I am also happy to report that my HA1c (diabetes test) was in the normal range! Yes, normal, not even prediabetic! So, it is possible to mange Type II diabetes without drugs. I typed this on an iPad, so expect errors. I probably made a few mistakes regarding times too, so expect a few tweaks here and there by the end of the day.- 7 replies
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Eur J Gastroenterol Hepatol. 2002 Apr;14(4):425-7. Related Articles, Links Celiac.com 07/30/2004 - The following abstract of a study that was done in 2002 emphasizes the importance of vitamin supplementation in the treatment of many celiacs: Dickey W. - Department of Gastroenterology, Altnagelvin Hospital, Londonderry BT47 6SB, Northern Ireland. OBJECTIVE: Although coeliac disease is a disorder of the proximal small bowel, associated vitamin B12 deficiency has been reported. This study aimed to assess the prevalence of B12 deficiency in a large series of coeliac patients, and to exclude the possibility that it is due to associated autoimmune gastritis. DESIGN: Prospective routine measurement of serum B12 in coeliac patients, with investigations for pernicious anaemia/autoimmune gastritis in B12-deficient patients. SETTING: Gastroenterology department of a large district general hospital. INTERVENTIONS: If they were not taking vitamin B12 supplements already, patients had serum B12 measured before starting dietary gluten exclusion. Those with low levels also had gastric biopsies taken and plasma gastrin and serum gastric parietal cell and intrinsic factor antibodies measured. MAIN OUTCOME MEASURES: Prevalence of low serum B12, and presence or absence of indicators of pernicious anaemia/autoimmune gastritis in patients with low serum B12. RESULTS: Of 159 patients, 13 had low serum B12 at diagnosis. A further six had been receiving B12 replacement therapy for 3-37 years before diagnosis, giving an overall prevalence of 12% (19 patients). Only 2/19 patients had gastric corpus atrophy, one with intrinsic factor antibodies and the other with hypergastrinaemia. There was no relationship between low B12 and clinical characteristics. CONCLUSIONS: Low B12 is common in coeliac disease without concurrent pernicious anaemia, and may be a presenting manifestation. B12 status should be known before folic acid replacement is started.
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