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- Celiac Disease & Gluten Intolerance Research
Celiac Disease & Gluten Intolerance Research
Growing evidence suggests that long noncoding RNAs (lncRNAs) play an important role in gene expression, especially that which influences inflammation.
What's ultra-short celiac disease, and what sets it apart from standard celiac disease?
New guidelines reverse previous recommendations on infant gluten introduction to prevent celiac disease. What's going on?
Can predictive values of transglutaminase (tTG) antibodies help diagnose celiac disease in kids, without the need for a biopsy?
Dutch researchers recently completed a large study of celiac patients based on symptoms, co-occurrence of immune mediated diseases, and malignancies.
A team of researchers recently completed the first extensive study comparing gene expression in children and adults with celiac disease, and found some key differences between the two groups.
Women and girls who have Turner syndrome are significantly more likely to have celiac disease than those without the sex chromosome anomaly, according to a new study by Scandinavian researchers.
How come only 2% to 5% of genetically susceptible individuals develop celiac disease? Gut microbes may be the key.
Does a lone protein in the gut trigger the inflammation and discomfort associated with gluten-sensitivity in people without celiac disease?
Laboratory tests for hemoglobin, ferritin, calcium, folate, vitamin B12, vitamin D, and thyroid function are regularly ordered in children with celiac disease, despite sufficient evidence for their necessity. To determine the frequency of nutritional deficiencies and levels of thyroid dysfunction in children with celiac disease, researches conducted a study that examined children before and after the initiation of a gluten-free diet.
Could gluten immunogenic peptides tell doctors how closely you've been following a gluten-free diet, and how well your gut is healing?
Recently, several studies have set out to determine how intake of gluten during infancy influences later risk of celiac disease.
A new study by researchers in Italy shows that only a minority of patients who meet clinical criteria for non-celiac gluten sensitivity actually show symptoms when exposed to gluten in a controlled gluten challenge. Why is that?
Has a Canadian researcher discovered a big clue toward preventing celiac disease?
A new study clarifies the celiac risk for close relatives of people with the disease.
If you thought celiac disease was slippery, try studying non-celiac gluten sensitivity.
Can mass screening for celiac disease help enough people, and improve enough lives to justify the cost and effort?
A research team recently conducted an analysis of the relationship between seronegative celiac disease (SNCD) and immunoglobulin deficiencies.
According to the latest press release, aspergillus niger prolyl endoprotease (AN-PEP) efficiently degrades gluten molecules into non-immunogenic peptides. But so what?
What can an isolated tribe of indigenous South Americans who have only recently begun eating wheat tell researchers about celiac disease?